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Thomas M. Badger

Researcher at University of Arkansas for Medical Sciences

Publications -  305
Citations -  13313

Thomas M. Badger is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Soy protein & Offspring. The author has an hindex of 63, co-authored 299 publications receiving 12304 citations. Previous affiliations of Thomas M. Badger include University of Arkansas & United States Department of Agriculture.

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Ethanol Induction of Class I Alcohol Dehydrogenase Expression in the Rat Occurs through Alterations in CCAAT/Enhancer Binding Proteins β and γ

TL;DR: These data provide the first evidence for the mechanism by which ethanol regulates rat hepatic Class I ADH gene expression in vivo, which involves the C/EBP site and the enhancer binding proteins β and γ.
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Alcoholic Liver Disease in Rats Fed Ethanol as Part of Oral or Intragastric Low-Carbohydrate Liquid Diets

TL;DR: Oral low- carbohydrate diets produce more ethanol-induced liver pathology than oral high-carbohydrate diets, but hepatotoxicity is more severe when a low-carb carbohydrate diet plus ethanol is infused intragastrically and is accompanied by significant increases in levels of proinflammatory cytokines.
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Cytotoxic hydroxylated triterpene alcohol ferulates from rice bran.

TL;DR: Three hydroxylated triterpene alcohol ferulates, (24S)-cycloart-25-ene-3 Beta,24-diol-3 beta-trans-ferulate, and (24R) cycloart -23Z-ene -3 beta,25- diol- 3 beta-Trans-ferulates, along with known compounds cycloartenol trans-ferulating and 24-methylenecycloartanol trans- ferulate were
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Early Infant Diet and the Omega 3 Fatty Acid DHA: Effects on Resting Cardiovascular Activity and Behavioral Development During the First Half-Year of Life

TL;DR: In infants fed the DHA-deficient diet, higher HR and lower values for heart rate variability measures were observed, indicating decreased parasympathetic tone in this group, and the absence of these effects in SF infants receiving the D HA-supplemented formula suggests that neither soy protein nor the associated phytochemicals in soy formula contribute to these effects to any appreciable extent.
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Vitamin D Supplementation Protects against Bone Loss Associated with Chronic Alcohol Administration in Female Mice

TL;DR: Dietary VitD supplementation may prevent skeletal toxicity in chronic drinkers by normalizing calcium homeostasis, preventing apoptosis, and suppressing EtOH-induced increases in bone resorption.