T
Thomas M. Badger
Researcher at University of Arkansas for Medical Sciences
Publications - 305
Citations - 13313
Thomas M. Badger is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Soy protein & Offspring. The author has an hindex of 63, co-authored 299 publications receiving 12304 citations. Previous affiliations of Thomas M. Badger include University of Arkansas & United States Department of Agriculture.
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Journal ArticleDOI
RNA-seq analysis of the rat placentation site reveals maternal obesity-associated changes in placental and offspring thyroid hormone signaling
Jessica Saben,Ping Kang,Ying Zhong,Keshari M. Thakali,Horacio Gomez-Acevedo,Sarah J. Borengasser,Aline Andres,Thomas M. Badger,Kartik Shankar +8 more
TL;DR: Gene expression analysis of placenta and offspring revealed that each utero-placental compartment responds distinctly to maternal OB with changes in inflammatory signaling, lipid metabolism and hormone stimulus being the predominant effects.
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p47phox-Nox2-dependent ROS Signaling Inhibits Early Bone Development in Mice but Protects against Skeletal Aging
Jin-Ran Chen,Oxana P. Lazarenko,Michael L. Blackburn,Kelly E. Mercer,Thomas M. Badger,Martin J. J. Ronis +5 more
TL;DR: It is indicated that the observed age-related switch of bone mass in p47phox-deficient mice occurs through an increased inflammatory milieu in bone and that p47PHox-Nox2-dependent physiological ROS signaling suppresses inflammation in aging.
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Flow cytometric analysis of lymphocytes from rats following chronic ethanol treatment
TL;DR: These data are the first to demonstrate that chronic alcohol intake alters lymphoid cell populations in the peripheral blood and primary organs of the immune systems in the presence of adequate nutrition.
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QMR: validation of an infant and children body composition instrument using piglets against chemical analysis.
TL;DR: QMR provides precise and accurate measures of FM, FFM and TBW in piglets weighing up to 50 kg and suggests that QMR should provide the opportunity to acquire valuable body composition data in longitudinal studies in children, which is not possible or practical with other commercially available instrumentation.
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Chronic ethanol exposure is associated with a local increase in TNF-α and decreased proliferation in the rat distraction gap
Daniel S. Perrien,Zhendong Liu,Elizabeth C. Wahl,Robert C. Bunn,Robert A. Skinner,James Aronson,James Aronson,John L. Fowlkes,Thomas M. Badger,Charles K. Lumpkin,Charles K. Lumpkin +10 more
TL;DR: The hypothesis that attenuation of bone formation by ethanol may be mediated, in part, by local increases in TNF-alpha during osteogenesis is supported.