scispace - formally typeset
T

Thomas M. Badger

Researcher at University of Arkansas for Medical Sciences

Publications -  305
Citations -  13313

Thomas M. Badger is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Soy protein & Offspring. The author has an hindex of 63, co-authored 299 publications receiving 12304 citations. Previous affiliations of Thomas M. Badger include University of Arkansas & United States Department of Agriculture.

Papers
More filters
Journal ArticleDOI

Carbohydrate deficiency as a possible factor in ethanol‐induced hepatic necrosis

Thomas M. Badger, +6 more
- 01 May 1998 - 
Journal ArticleDOI

Growth hormone-regulated periportal expression of CYP2C7 in rat liver

Teija Oinonen, +5 more
- 01 Mar 2000 - 
TL;DR: It is demonstrated that growth hormone up-regulates the CYP2C7 gene by enhancing the expression of the protein specifically in the periportal liver region, and growth hormone may up- Regulate other periportally expressed liver genes in a similar fashion.
Journal ArticleDOI

Estrogenic status modulates the effect of soy on hepatic responses to 7,12-dimethylbenz(a)anthracene (DMBA)

Rohit Singhal, +2 more
- 01 Jan 2009 - 
TL;DR: The data suggest that although E2 status does not effect soy-mediated AhR degradation, it modulates the effects of soy on many genes, including CYP1A1.
Journal ArticleDOI

Resting gamma power during the postnatal critical period for GABAergic system development is modulated by infant diet and sex.

R.T. Pivik, +8 more
- 01 Jan 2019 - 
TL;DR: The results show gamma power follows a gradually increasing function across this time period that varies in topographic magnitude and is differentially influenced by subject and environmental variables-among which gestation, head circumference, and infant diet-sex interactions figure most prominently.
Journal ArticleDOI

Global Deletion of Glutathione S-Transferase A4 Exacerbates Developmental Nonalcoholic Steatohepatitis.

Martin J. J. Ronis, +10 more
- 01 Feb 2017 - 
TL;DR: The data suggest that lipid peroxidation products play a role in progression of liver injury to steatohepatitis in NASH produced by high-fat feeding during development but appear less important in development of fibrosis.