T
Thomas M. Badger
Researcher at University of Arkansas for Medical Sciences
Publications - 305
Citations - 13313
Thomas M. Badger is an academic researcher from University of Arkansas for Medical Sciences. The author has contributed to research in topics: Soy protein & Offspring. The author has an hindex of 63, co-authored 299 publications receiving 12304 citations. Previous affiliations of Thomas M. Badger include University of Arkansas & United States Department of Agriculture.
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Journal ArticleDOI
Loss of functional NADPH oxidase 2 protects against alcohol-induced bone resorption in female p47phox-/- mice.
Kelly E. Mercer,Clark R. Sims,Carrie Yang,Rebecca Wynne,Christopher Moutos,William R. Hogue,Charles K. Lumpkin,Larry J. Suva,Jin-Ran Chen,Thomas M. Badger,Martin J. J. Ronis +10 more
TL;DR: In osteoblasts, NOX2 and NOX4 appear to work in tandem to increase RANKL expression, whereas EtOH-mediated inhibition of bone formation occurs via a NoX2-independent mechanism, which suggests that NOx2-derived ROS is necessary for Etoh-induced bone resorption.
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Formula feeding alters hepatic gene expression signature, iron and cholesterol homeostasis in the neonatal pig
Martin J. J. Ronis,Ying Chen,Kartik Shankar,Horatio Gomez-Acevedo,Mario A. Cleves,Jamie Badeaux,Michael L. Blackburn,Thomas M. Badger +7 more
TL;DR: In this article, neonatal piglets were breast fed and compared with those fed commercially available milk-based formula (mixture of formula and breast milk), and the results showed that formula feeding remains more popular than breast-feeding.
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Cysteamine Induces Depletion of Both Immunological and Biological Prolactin Activity in the Anterior Pituitary and Blood of the Rat
William J. Millard,Stephen M. Sagar,James W. Simpkins,Robert E. Owens,Thomas M. Badger,Henry G. Friesen,Joseph B. Martin +6 more
TL;DR: CSH or similar compounds may serve as a prototype for a new class of drugs which can be used in the treatment of hyperprolactinemia and circumvents the DA receptor to deplete pituitary PRL content.
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Distinct adipogenic differentiation phenotypes of human umbilical cord mesenchymal cells dependent on adipogenic conditions.
Jessica Saben,Keshari M. Thakali,Forrest Lindsey,Ying Zhong,Thomas M. Badger,Aline Andres,Kartik Shankar +6 more
TL;DR: The results suggest that insulin signaling pathways of differentiated UCMSCs are functionally similar to adipocytes, and the presence of indomethacin greatly enhances their adipogenic potential beyond that of rosiglitazone.
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Regulation of the hepatic CYP 2E1 gene during chronic alcohol exposure: lack of an ethanol response element in the proximal 5'-flanking sequence.
TL;DR: It is demonstrated that bacterially expressed H NF-1alpha in footprint assays bind Site C sequences and that HNF-1 alpha transactivates the CYP 2E1 promoter in co-transfection experiments with HNF -1alpha expression plasmid and plasmids containing CYP2E1 promoters sequences coupled to the chloramphenicol acetyl transferase gene.