scispace - formally typeset
T

Tomasz Urbanek

Researcher at Medical University of Silesia

Publications -  123
Citations -  1491

Tomasz Urbanek is an academic researcher from Medical University of Silesia. The author has contributed to research in topics: Deep vein & Varicose veins. The author has an hindex of 18, co-authored 122 publications receiving 1048 citations. Previous affiliations of Tomasz Urbanek include Oslo University Hospital & University of Silesia in Katowice.

Papers
More filters
Journal ArticleDOI

The 2020 update of the CEAP classification system and reporting standards

TL;DR: The CEAP Task Force has adopted the revised Delphi process and made several changes, including adding Corona phlebectatica as the C4c clinical subclass, introducing the modifier "r" for recurrent varicose veins and recurrent venous ulcers, and replacing numeric descriptions of the venous segments by their common abbreviations.
Journal ArticleDOI

A comparative clinical study on five types of compression therapy in patients with venous leg ulcers.

TL;DR: Analysis of changes of the percentage of Gilman index and wound total surface area confirmed that intermittent pneumatic compression, stockings and multi layer bandages are the most efficient in venous leg ulcers.
Journal Article

Venous hemodynamic changes in lower limb venous disease : the UIP consensus according to scientific evidence

TL;DR: The aim was to confirm or dispel long-held hemodynamic principles and to provide a comprehensive review of venous hemodynamic concepts underlying the pathophysiology of lower limb venous disorders, their usefulness for investigating patients and the relevant hemodynamic changes associated with various forms of treatment.
Journal ArticleDOI

Rapamycin induces of protective autophagy in vascular endothelial cells exposed to oxygen-glucose deprivation.

TL;DR: It is demonstrated that toxicity of simulated ischemia conditions were enhanced in HUVEC when autophagy was blocked, and that rapamycin effectively prevented OGD-evoked damage by induction of protective autophagic via inhibition of the mTOR pathway.