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Xiangyu Deng
Researcher at Huazhong University of Science and Technology
Publications - 34
Citations - 866
Xiangyu Deng is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Protein kinase B & PI3K/AKT/mTOR pathway. The author has an hindex of 13, co-authored 30 publications receiving 399 citations. Previous affiliations of Xiangyu Deng include Nanyang Technological University.
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Solutions to the Drawbacks of Photothermal and Photodynamic Cancer Therapy
TL;DR: In this article, the problems and shortcomings encountered in the development of phototherapy and highlights possible solutions to address them so that phototherapy may become a useful cancer treatment approach in clinical practice.
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Mechanisms of endogenous repair failure during intervertebral disc degeneration
TL;DR: The current research situation and progress of the mechanisms through which IVD stem/progenitor cells failed to repair IVD tissues during IVD degeneration are reviewed to provide an innovative research direction for endogenous repair and a new potential treatment strategy for IVd degeneration.
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Accelerated Bone Regeneration by Gold-Nanoparticle-Loaded Mesoporous Silica through Stimulating Immunomodulation.
Hang Liang,Chen Jin,Liang Ma,Xiaobo Feng,Xiangyu Deng,Shuilin Wu,Shuilin Wu,Xiangmei Liu,Cao Yang +8 more
TL;DR: It is demonstrated that gold nanoparticle-loaded mesoporous silica nanoparticles (Au-MSNs) could significantly promote osteogenic activity by modulating the immune microenvironment, showing its therapeutic potential for bone tissue repair and regeneration.
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Polarization and function of tumor-associated macrophages mediate graphene oxide-induced photothermal cancer therapy.
TL;DR: This study identified PTT treatment as an approach for preventing osteosarcoma invasion by inhibition of M2 polarization by using graphene oxide combined with polyethylene glycol as the photothermal material to induce heating effect in macrophages to define its anti-tumor effect in vitro and in vivo.
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Mesenchymal Stem Cells Protect Nucleus Pulposus Cells from Compression-Induced Apoptosis by Inhibiting the Mitochondrial Pathway
TL;DR: It is suggested that BMSCs can protect against compression-induced apoptosis of NPCs by inhibiting the mitochondrial pathway and thus enhance the understanding on the MSC-based therapy for IVDD.