Y
Yi Li
Researcher at Boston Children's Hospital
Publications - 20
Citations - 1828
Yi Li is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 14, co-authored 15 publications receiving 1381 citations. Previous affiliations of Yi Li include Harvard University & Mayo Clinic.
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Journal ArticleDOI
Energy metabolism of rabbit retina as related to function: high cost of Na+ transport
TL;DR: A surprisingly large fraction of oxidative energy could not be assigned to phototransduction, to neurotransmission, to Na+ transport for other purposes, or to vegetative metabolism, and may be related to the (previously reported) rapid turnover of the gamma-phosphate of retinal GTP.
Journal ArticleDOI
New mammalian chloride channel identified by expression cloning.
Markus Paulmichl,Markus Paulmichl,Yi Li,Yi Li,Kevin Wickman,Michael J. Ackerman,Ernest G. Peralta,Ernest G. Peralta,David E. Clapham +8 more
TL;DR: A complementary DNA from an epithelial cell line has been isolated, sequenced and its putative structure examined, suggesting a new class of ion channels not related to other previously cloned chloride channels.
Journal ArticleDOI
Reactivation of Dormant Relay Pathways in Injured Spinal Cord by KCC2 Manipulations
Bo Chen,Yi Li,Bin Yu,Zicong Zhang,Benedikt Brommer,Philip R. Williams,Yuanyuan Liu,Shane V. Hegarty,Songlin Zhou,Junjie Zhu,Hong Guo,Yi Lu,Yiming Zhang,Xiaosong Gu,Zhigang He +14 more
TL;DR: Spinal inhibitory interneurons are identified as a roadblock limiting the integration of descending inputs into relay circuits after injury and KCC2 agonists are suggested as promising treatments for promoting functional recovery after spinal cord injury.
Journal ArticleDOI
Generating yeast transcriptional activators containing no yeast protein sequences.
TL;DR: A repeat of the original experiment using the complete Lex A molecule LexA(1–202) as the DNA-binding region, instead of GAL4 or LexA (1–87), finds that about 1% of the short peptides encoded by E. coli genomic fragments act as transcriptional activating regions when fused to intact LexA.
Journal ArticleDOI
Microglia-organized scar-free spinal cord repair in neonatal mice.
Yi Li,Yi Li,Xuelian He,Xuelian He,Riki Kawaguchi,Riki Kawaguchi,Yu Zhang,Yu Zhang,Q. Wang,Q. Wang,Aboozar Monavarfeshani,Aboozar Monavarfeshani,Zhiyun Yang,Zhiyun Yang,Bo Chen,Bo Chen,Zhongju Shi,Zhongju Shi,Huyan Meng,Huyan Meng,Songlin Zhou,Songlin Zhou,Junjie Zhu,Junjie Zhu,Anne Jacobi,Anne Jacobi,Vivek Swarup,Phillip G. Popovich,Daniel H. Geschwind,Daniel H. Geschwind,Zhigang He,Zhigang He +31 more
TL;DR: The cellular and molecular basis of the nearly complete recovery of neonatal mice after spinal cord injury is revealed, and strategies that could be used to facilitate scar-free healing in the adult mammalian nervous system are suggested.