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Yul Eum Song

Researcher at University of Missouri

Publications -  7
Citations -  317

Yul Eum Song is an academic researcher from University of Missouri. The author has contributed to research in topics: Incineration & Food waste. The author has an hindex of 5, co-authored 7 publications receiving 244 citations. Previous affiliations of Yul Eum Song include St. Jude Children's Research Hospital & Dongguk University.

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Evaluation of food waste disposal options by LCC analysis from the perspective of global warming: Jungnang case, South Korea.

TL;DR: The costs associated with eight food waste disposal options, dry feeding, wet feeding, composting, anaerobic digestion, co-digestion with sewage sludge, food waste disposer, incineration, and landfilling, were evaluated in the perspective of global warming and energy and/or resource recovery.
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A lipid-based partitioning mechanism for selective incorporation of proteins into membranes of HIV particles.

TL;DR: It is shown that protein sorting into the membranes of budding HIV particles is dependent on lipid-based partitioning of proteins, through a mechanism initiated by HIV protein Gag oligomerization at viral assembly sites.
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Evaluation of food waste disposal options in terms of global warming and energy recovery: Korea

TL;DR: In this article, the authors evaluate and compare environmental impacts of various food waste management systems: anaerobic digestion, co-digestion with sewage sludge, and volume reduction using a garbage dryer followed by incineration from generation to final disposal.
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Sphingosine 1-Phosphate Lyase Enhances the Activation of IKKε To Promote Type I IFN-Mediated Innate Immune Responses to Influenza A Virus Infection.

TL;DR: It is demonstrated that SPL functions as a positive regulator of IKKε to propel type I IFN–mediated innate immune responses against viral infection and the functional significance of the SPL–IKKε–IFN axis during host innate immunity against viral infections is illustrated.
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βTrCP is Required for HIV-1 Vpu Modulation of CD4, GaLV Env, and BST-2/Tetherin.

TL;DR: The data suggest that the primary role of the S52,56 residues in antagonism of CD4, GaLV Env, and BST-2/tetherin is to recruit the SCF/βTrCP ubiquitin ligase.