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Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format
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Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format Example of Virus Genes format
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open access Open Access ISSN: 9208569 e-ISSN: 1572994X

Virus Genes — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Genetics #177 of 325 up up by 21 ranks
Virology #41 of 69 up up by 3 ranks
Molecular Biology #260 of 382 up up by 15 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 399 Published Papers | 1507 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 07/06/2020
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FAQ

Journal Performance & Insights

  • Impact Factor
  • CiteRatio
  • SJR
  • SNIP

Impact factor determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

1.991

23% from 2018

Impact factor for Virus Genes from 2016 - 2019
Year Value
2019 1.991
2018 1.616
2017 1.542
2016 1.431
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 23% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

3.8

12% from 2019

CiteRatio for Virus Genes from 2016 - 2020
Year Value
2020 3.8
2019 3.4
2018 3.1
2017 3.0
2016 2.9
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has increased by 12% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

0.642

12% from 2019

SJR for Virus Genes from 2016 - 2020
Year Value
2020 0.642
2019 0.732
2018 0.655
2017 0.711
2016 0.653
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 12% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

0.694

5% from 2019

SNIP for Virus Genes from 2016 - 2020
Year Value
2020 0.694
2019 0.727
2018 0.606
2017 0.749
2016 0.687
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has decreased by 5% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

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Virus Genes

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Springer

Virus Genes

VIRUS GENES publishes studies on analysis of virus genes, gene products and functions, regulation of virus gene function, cell biology of virus infection functional studies of genes and gene families, encoded by eukaryotic,  prokaryotic and archaeal viruses, viroids, as well a...... Read More

Medicine

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Last updated on
07 Jun 2020
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ISSN
0920-8569
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Impact Factor
Medium - 0.92
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
SPBASIC
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Citation Type
Author Year
(Blonder et al, 1982)
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Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1023/A:1007989407305
Origin and evolution of viruses
John J. Holland1, Esteban Domingo2
01 Jan 1998 - Virus Genes

Abstract:

P. Schuster and P.F. Stadler, Nature and Evolution of Early Replicons. H.D. Robertson and O.D. Neel, Virus Origins: Conjoined RNA Genomes as Precursors to DNA Genomes. J.S. Semancik and N. Duran-Vila, Viroids in Plants: Shadows and Footprints of a Primitive RNA. C. Biebricher, Mutation, Competition, and Selection as Measured ... P. Schuster and P.F. Stadler, Nature and Evolution of Early Replicons. H.D. Robertson and O.D. Neel, Virus Origins: Conjoined RNA Genomes as Precursors to DNA Genomes. J.S. Semancik and N. Duran-Vila, Viroids in Plants: Shadows and Footprints of a Primitive RNA. C. Biebricher, Mutation, Competition, and Selection as Measured with Small RNA Molecules. A. Meyerhans and J.-P. Vartanian, The Fidelity of Cellular and Viral Polymerases and Its Manipulation for Hypermutagenesis. S. Wain-Hobson and M. Sala, Drift and Conservatism in RNA Virus Evolution: Are They Adapting or Merely Changing? E. Domingo, C. Escarmis, L. Menendez-Aarias, and J.J. Holland, Viral Quasispecies and Fitness Variations. M.A. McClure, The Retroid Agents: Disease, Function, and Evolution. D. Wodarz and M.A. Nowak, Dynamics of HIV Pathogenesis and Treatment. I.M. Rouzine and J.M. Coffin, Interplay between Experiment and Theory in Development of a Working Model for HIV-1 Population Dynamics. A.J. Gibbs, P.L. Keese, M.J. Gibbs, and F. Garcia-Arenal, Plant Virus Evolution: Past, Present, and Future. M. Gromeier, E. Wimmer, and A.E. Gorbalenya, Genetics, Pathogenesis, and Evolution of Picornaviruses. J.I. Esteban, M. Martell, W.F. Carman, and J. Gomez, The Impact of Rapid Evolution of the Hepatitis Viruses. R.G. Webster, Antigenic Variation in Influenza Viruses. L.P. Villarreal, DNA Virus Contribution to Host Evolution. C.R. Parrish and U. Truyen, Parvovirus Variation and Evolution. D.J. McGeoch and A.J. Davison, The Molecular Evolutionary History of the Herpesviruses. J. Salas, M.L. Salas, and E. Vinuela, African Swine Fever Virus: A Missing Link Between Poxviruses and Iridoviruses? Subject Index. read more read less

Topics:

Viral evolution (62%)62% related to the paper, RNA virus (57%)57% related to the paper, Viral quasispecies (54%)54% related to the paper, DNA virus (52%)52% related to the paper, Population (52%)52% related to the paper
556 Citations
open accessOpen access Journal Article DOI: 10.1007/S11262-012-0713-1
Porcine epidemic diarrhoea virus: a comprehensive review of molecular epidemiology, diagnosis, and vaccines.
Daesub Song1, Bong-Kyun Park2
22 Jan 2012 - Virus Genes

Abstract:

The porcine epidemic diarrhoea virus (PEDV), a member of the Coronaviridae family, causes acute diarrhoea and dehydration in pigs. Although it was first identified in Europe, it has become increasingly problematic in many Asian countries, including Korea, China, Japan, the Philippines, and Thailand. The economic impacts of th... The porcine epidemic diarrhoea virus (PEDV), a member of the Coronaviridae family, causes acute diarrhoea and dehydration in pigs. Although it was first identified in Europe, it has become increasingly problematic in many Asian countries, including Korea, China, Japan, the Philippines, and Thailand. The economic impacts of the PEDV are substantial, given that it results in significant morbidity and mortality in neonatal piglets and is associated with increased costs related to vaccination and disinfection. Recently, progress has been made in understanding the molecular epidemiology of PEDV, thereby leading to the development of new vaccines. In the current review, we first describe the molecular and genetic characteristics of the PEDV. Then we discuss its molecular epidemiology and diagnosis, what vaccines are available, and how PEDV can be treated. read more read less

Topics:

Porcine epidemic diarrhea virus (61%)61% related to the paper, Porcine epidemic diarrhoea (61%)61% related to the paper, Molecular epidemiology (52%)52% related to the paper
View PDF
494 Citations
Journal Article DOI: 10.1023/A:1024455415443
The evolution, distribution and diversity of endogenous retroviruses.
Robert J. Gifford1, Michael Tristem1
01 May 2003 - Virus Genes

Abstract:

The retroviral capacity for integration into the host genome can give rise to endogenous retroviruses (ERVs): retroviral sequences that are transmitted vertically as part of the host germ line, within which they may continue to replicate and evolve ERVs represent both a unique archive of ancient viral sequence information and... The retroviral capacity for integration into the host genome can give rise to endogenous retroviruses (ERVs): retroviral sequences that are transmitted vertically as part of the host germ line, within which they may continue to replicate and evolve ERVs represent both a unique archive of ancient viral sequence information and a dynamic component of host genomes As such they hold great potential as informative markers for studies of both virus evolution and host genome evolution Numerous novel ERVs have been described in recent years, particularly as genome sequencing projects have advanced This review discusses the evolution of ERV lineages, considering the processes by which ERV distribution and diversity is generated The diversity of ERVs isolated so far is summarised in terms of both their distribution across host taxa, and their relationships to recognised retroviral genera Finally the relevance of ERVs to studies of genome evolution, host disease and viral ecology is considered, and recent findings discussed read more read less

Topics:

Paleovirology (63%)63% related to the paper, Endogenous retrovirus (63%)63% related to the paper, Genome evolution (56%)56% related to the paper, Viral evolution (53%)53% related to the paper, Genome (50%)50% related to the paper
331 Citations
Journal Article DOI: 10.1007/S11262-009-0412-8
The biological properties of E6 and E7 oncoproteins from human papillomaviruses
Raffaella Ghittoni1, Rosita Accardi1, Uzma Hasan1, Tarik Gheit1, Bakary S. Sylla1, Massimo Tommasino1
01 Feb 2010 - Virus Genes

Abstract:

More than 100 different human papillomavirus (HPV) types have been isolated so far, and they can be sub-grouped in cutaneous or mucosal according to their ability to infect the skin or the mucosa of the genital or upper-respiratory tracts. A sub-group of human mucosal HPVs, referred to as high-risk HPV types, is responsible f... More than 100 different human papillomavirus (HPV) types have been isolated so far, and they can be sub-grouped in cutaneous or mucosal according to their ability to infect the skin or the mucosa of the genital or upper-respiratory tracts. A sub-group of human mucosal HPVs, referred to as high-risk HPV types, is responsible for approximately 5% of all human cancers, which represents one-third of all the tumours induced by viruses. Epidemiological and biological studies have shown that HPV16 is the most oncogenic type within the high-risk group. Emerging lines of evidence suggest that, in addition to the high-risk mucosal HPV types, certain cutaneous HPVs are involved in skin cancer. HPV-associated cancers are intimately linked to HPV persistence and the accumulation of chromosomal rearrangements. The products of the early genes, E6 and E7, of the high-risk mucosal HPV types play a key role in both events. Indeed, these proteins have developed a number of strategies to evade host immuno-surveillance allowing viral persistence, and to alter cell cycle and apoptosis control, facilitating the accumulation of DNA damage/mutations. Often, the two oncoproteins target the same cellular pathways with different mechanisms, showing a strong synergism in promoting cellular transformation and neutralizing the immune response. Here, we review most of the findings on the biological properties and molecular mechanisms of the oncoproteins E6 and E7 from mucosal and cutaneous HPV types. read more read less
272 Citations
open accessOpen access Journal Article DOI: 10.1023/A:1011831902219
Completion of the porcine epidemic diarrhoea coronavirus (PEDV) genome sequence.
Rolf Kocherhans1, Anne Bridgen2, Mathias Ackermann1, Kurt Tobler
01 Jan 2001 - Virus Genes

Abstract:

The sequence of the replicase gene of porcine epidemic diarrhoea virus (PEDV) has been determined. This completes the sequence of the entire genome of strain CV777, which was found to be 28,033 nucleotides (nt) in length (excluding the poly A-tail). A cloning strategy, which involves primers based on conserved regions in the ... The sequence of the replicase gene of porcine epidemic diarrhoea virus (PEDV) has been determined. This completes the sequence of the entire genome of strain CV777, which was found to be 28,033 nucleotides (nt) in length (excluding the poly A-tail). A cloning strategy, which involves primers based on conserved regions in the predicted ORF1 products from other coronaviruses whose genome sequence has been determined, was used to amplify the equivalent, but as yet unknown, sequence of PEDV. Primary sequences derived from these products were used to design additional primers resulting in the amplification and sequencing of the entire ORF1 of PEDV. Analysis of the nucleotide sequences revealed a small open reading frame (ORF) located near the 5′ end (no 99–137), and two large, slightly overlapping ORFs, ORF1a (nt 297–12650) and ORF1b (nt 12605–20641). The ORF1a and ORF1b sequences overlapped at a potential ribosomal frame shift site. The amino acid sequence analysis suggested the presence of several functional motifs within the putative ORF1 protein. By analogy to other coronavirus replicase gene products, three protease and one growth factor-like motif were seen in ORF1a, and one polymerase domain, one metal ion-binding domain, and one helicase motif could be assigned within ORF1b. Comparative amino acid sequence alignments revealed that PEDV is most closely related to human coronavirus (HCoV)-229E and transmissible gastroenteritis virus (TGEV) and less related to murine hepatitis virus (MHV) and infectious bronchitis virus (IBV). These results thus confirm and extend the findings from sequence analysis of the structural genes of PEDV. read more read less

Topics:

Porcine epidemic diarrhea virus (60%)60% related to the paper, Sequence analysis (58%)58% related to the paper, Coronavirus (57%)57% related to the paper, Peptide sequence (54%)54% related to the paper, Porcine epidemic diarrhoea (51%)51% related to the paper
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230 Citations
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Virus Genes format uses SPBASIC citation style.

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SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the Virus Genes guidelines and autoformat it.

Yes. The template is fully compliant as per the guidelines of this journal. Our experts at SciSpace ensure that. Also, if there's any update in the journal format guidelines, we take care of it and include that in our algorithm.

Sure. We support all the top citation styles like APA style, MLA style, Vancouver style, Harvard style, Chicago style, etc. For example, in case of this journal, when you write your paper and hit autoformat, it will automatically update your article as per the Virus Genes citation style.

You can avail our Free Trial for 7 days. I'm sure you'll find our features very helpful. Plus, it's quite inexpensive.

Yup. You can choose the right template, copy-paste the contents from the word doc and click on auto-format. You'll have a publish-ready paper that you can download at the end.

A matter of seconds. Besides that, our intuitive editor saves a load of your time in writing and formating your manuscript.

One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per Virus Genes's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

Absolutely! You can do it using our intuitive editor. It's very easy. If you need help, you can always contact our support team.

SciSpace is an online tool for now. We'll soon release a desktop version. You can also request (or upvote) any feature that you think might be helpful for you and the research community in the feature request section once you sign-up with us.

Sure. You can request any template and we'll have it up and running within a matter of 3 working days. You can find the request box in the Journal Gallery on the right sidebar under the heading, "Couldn't find the format you were looking for?".

After you have written and autoformatted your paper, you can download it in multiple formats, viz., PDF, Docx and LaTeX.

To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

The 5 most common citation types in order of usage are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

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After uploading your paper on SciSpace, you would see a button to request a journal submission service for Virus Genes.

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Yes. SciSpace provides this functionality.

After signing up, you would need to import your existing references from Word or .bib file.

SciSpace would allow download of your references in Virus Genes Endnote style, according to springer guidelines.

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