Journal ArticleDOI
Effects of chronic exercise and deconditioning on platelet function in women
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It is suggested that training-induced platelet functional changes in women in the midfollicular phase may be mediated by nitric oxide.Abstract:
Wang, Jong-Shyan, Chauying J. Jen, and Hsiun-Ing Chen.Effects of chronic exercise and deconditioning on platelet function in women. J. Appl. Physiol.83(6): 2080–2085, 1997.—To investigate the effec...read more
Citations
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Exercise and Hypertension
Linda S. Pescatello,Barry A. Franklin,Robert Fagard,William B. Farquhar,George A. Kelley,Chester A. Ray +5 more
TL;DR: Exercise remains a cornerstone therapy for the primary prevention, treatment, and control of HTN and angiotensin converting enzyme (ACE) inhibitors and calcium channel blockers are currently the drugs of choice for recreational exercisers and athletes who have HTN.
Journal ArticleDOI
Effect of aerobic exercise on blood pressure: a meta-analysis of randomized, controlled trials.
TL;DR: A meta-analysis of 54 trials showed that previously sed-entary adults could decrease systolic blood pressure with aerobic exercise and showed smaller reduc-tions in blood pressure in trials lasting longer than 6 months, perhaps because of difficulties in sustaining regular exercise.
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Effect of Aerobic Exercise on Blood Pressure
TL;DR: In this article, an increase in aerobic physical activity should be considered an important component of lifestyle modification for pre-menopausal and postmenopausal individuals. But, it is not recommended for individuals with high blood pressure.
Journal ArticleDOI
Evidence for prescribing exercise as therapy in chronic disease.
TL;DR: The evidence for prescribing exercise therapy in the treatment of metabolic syndrome‐related disorders (insulin resistance, type 2 diabetes, dyslipidemia, hypertension, obesity), heart and pulmonary diseases, muscle, bone and joint diseases, and cancer, depression, asthma and type 1 diabetes is presented.
Journal ArticleDOI
Effects of Endurance Training on Blood Pressure, Blood Pressure–Regulating Mechanisms, and Cardiovascular Risk Factors
TL;DR: A aerobic endurance training decreases blood pressure through a reduction of vascular resistance, in which the sympathetic nervous system and the renin-angiotensin system appear to be involved, and favorably affects concomitant cardiovascular risk factors.
References
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Journal ArticleDOI
Analysis of nitrate, nitrite, and [15N]nitrate in biological fluids
Laura C. Green,David A. Wagner,Joseph A. Glogowski,Paul L. Skipper,John S. Wishnok,Steven R. Tannenbaum +5 more
TL;DR: A new automated system for the analysis of nitrate via reduction with a high-pressure cadmium column that automatically eliminates interference from other compounds normally present in urine and other biological fluids is described.
Journal ArticleDOI
Flow-induced release of endothelium-derived relaxing factor
TL;DR: It is demonstrated that in addition to prostacyclin, flow triggers the release of another relaxing substance (or substances) from vascular endothelial cells that has characteristics similar to the endothelium-derived relaxing factor released by acetylcholine.
Journal ArticleDOI
Menopause and Risk of Cardiovascular Disease: The Framingham Study
TL;DR: Although cholesterol and hemoglobin did rise somewhat more steeply in women undergoing the menopause, this greater incidence of cardiovascular disease in postmenopausal women could not be explained by the influence of theMenopause on the usual cardiovascular risk factors.
Journal ArticleDOI
Concurrent morning increase in platelet aggregability and the risk of myocardial infarction and sudden cardiac death.
Geoffrey H. Tofler,Damian Brezinski,Andrew I. Schafer,Charles A. Czeisler,John D. Rutherford,Stefan N. Willich,Ray E. Gleason,Gordon H. Williams,James E. Muller +8 more
TL;DR: The period from 6 to 9 a.m. was the only interval in the 24-hour period during which platelet aggregability increased significantly, and in vitro platelet responsiveness to either adenosine diphosphate or epinephrine was lower.
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Crucial role of endothelium in the vasodilator response to increased flow in vivo
TL;DR: Results suggest that endothelial cells act as mediators of flow-dependent dilation in vasomotor response to increases in flow as observed in conduit arteries.