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Open AccessJournal ArticleDOI

Eggs in Early Complementary Feeding and Child Growth: A Randomized Controlled Trial.

TLDR
It is shown that when offered early in complementary feeding, the food-based intervention of eggs produced a large effect size on child growth, supporting the hypothesis that early introduction of eggs significantly improved growth in young children.
Abstract
BACKGROUND: Eggs are a good source of nutrients for growth and development. We hypothesized that introducing eggs early during complementary feeding would improve child nutrition. METHODS: A randomized controlled trial was conducted in Cotopaxi Province, Ecuador, from March to December 2015. Children ages 6 to 9 months were randomly assigned to treatment (1 egg per day for 6 months [ n = 83]) and control (no intervention [ n = 80]) groups. Both arms received social marketing messages to encourage participation in the Lulun Project ( lulun meaning “egg” in Kichwa). All households were visited once per week to monitor morbidity symptoms, distribute eggs, and monitor egg intakes (for egg group only). Baseline and end point outcome measures included anthropometry, dietary intake frequencies, and morbidity symptoms. RESULTS: Mothers or other caregivers reported no allergic reactions to the eggs. Generalized linear regression modeling showed the egg intervention increased length-for-age z score by 0.63 (95% confidence interval [CI], 0.38–0.88) and weight-for-age z score by 0.61 (95% CI, 0.45–0.77). Log-binomial models with robust Poisson indicated a reduced prevalence of stunting by 47% (prevalence ratio [PR], 0.53; 95% CI, 0.37–0.77) and underweight by 74% (PR, 0.26; 95% CI, 0.10–0.70). Children in the treatment group had higher dietary intakes of eggs (PR, 1.57; 95% CI, 1.28–1.92) and reduced intake of sugar-sweetened foods (PR, 0.71; 95% CI, 0.51–0.97) compared with control. CONCLUSIONS: The findings supported our hypothesis that early introduction of eggs significantly improved growth in young children. Generally accessible to vulnerable groups, eggs have the potential to contribute to global targets to reduce stunting.

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References
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Journal ArticleDOI

Maternal and child undernutrition and overweight in low-income and middle-income countries

TL;DR: It is estimated that undernutrition in the aggregate--including fetal growth restriction, stunting, wasting, and deficiencies of vitamin A and zinc along with suboptimum breastfeeding--is a cause of 3·1 million child deaths annually or 45% of all child deaths in 2011.
Journal ArticleDOI

Evidence-based interventions for improvement of maternal and child nutrition: What can be done and at what cost?

TL;DR: In this article, the authors did a comprehensive update of interventions to address undernutrition and micronutrient deficiencies in women and children and used standard methods to assess emerging new evidence for delivery platforms.

Maternal and Child Nutrition 2 Evidence-based interventions for improvement of maternal and child nutrition: what can be done and at what cost?

TL;DR: Improved access for nutrition-sensitive approaches can greatly accelerate progress in countries with the highest burden of maternal and child undernutrition and mortality, if this improved access is linked to nutrition- sensitive approaches.
Journal ArticleDOI

Worldwide Timing of Growth Faltering: Revisiting Implications for Interventions

TL;DR: Comparison of child growth patterns in 54 countries with WHO standards shows that growth faltering in early childhood is even more pronounced than suggested by previous analyses based on the National Center for Health Statistics reference, confirming the need to scale up interventions during the window of opportunity defined by pregnancy and the first 2 years of life.
Journal ArticleDOI

Effects of Early Nutritional Interventions on the Development of Atopic Disease in Infants and Children: The Role of Maternal Dietary Restriction, Breastfeeding, Timing of Introduction of Complementary Foods, and Hydrolyzed Formulas

TL;DR: There is insufficient data to document a protective effect of any dietary intervention beyond 4 to 6 months of age for the development of atopic disease, and current evidence does not support a major role for maternal dietary restrictions during pregnancy or lactation.
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