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Journal ArticleDOI

Elevated plasma testosterone and gonadotropin levels in infertile males with hyperthyroidism.

Harrie R. Clyde, +2 more
- 01 Jun 1976 - 
- Vol. 27, Iss: 6, pp 662-666
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TLDR
In this article, the existence of male infertility in hyperthyroidism and the mechanisms adversely affecting spermatogenesis therein are discussed. But little attention has been given to the existence and mechanisms of these disorders.
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This article is published in Fertility and Sterility.The article was published on 1976-06-01. It has received 59 citations till now. The article focuses on the topics: Infertility & Male infertility.

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Citations
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Journal ArticleDOI

Thyroid Function and Human Reproductive Health

TL;DR: Through its interaction in several pathways, normal thyroid function is important to maintain normal reproduction and changes in SHBG and sex steroids are a consistent feature associated with hyper- and hypothyroidism and were already reported many years ago.
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Thyroid autoimmunity and hypothyroidism before and during pregnancy

TL;DR: In the present review, an attempt was made to describe current knowledge and concepts concerning the complex relationships that link thyroid autoimmunity (TAI) and hypothyroidism with female and male infertility, as well as abnormalities occurring during pregnancy, such as pregnancy loss and maternal and fetal repercussions associated with hypothyrogenism.
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Hormonal control of Sertoli cell metabolism regulates spermatogenesis

TL;DR: Sertoli cells (SCs) have functions that reach far beyond the physical support of germ cells, as they are responsible for creating the adequate ionic and metabolic environment for spermatogenesis as mentioned in this paper.
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Male reproductive function in relation with thyroid alterations.

TL;DR: The effects of hypothyroidism on male reproduction appear to be more subtle than those of hyperthyroidism and reversible, and Gonadal damage may be cumulative in those requiring multiple administrations and sperm banking should be considered in such patients.
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The Hypothalamic-Pituitary-Testicular Axis in Thyrotoxicosis*

TL;DR: The results suggest that men with hyperthyroidism have 1) partial Leydig cell failure, 2) impairment of spermatogenesis, and 3) blunting of the feedback effects of E2.
References
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Journal ArticleDOI

Hormonal control of mammalian spermatogenesis

TL;DR: Results from the studies indicate that changes in the metabolism of proteins lipids and carbohydrates take place in the testes under the control of hormones, but it may be that the observed biochemical changes are a reflection of the changes in cellular composition of testicular tissue rather than the result of metabolic changes within a specific cell type in response to the effect of a hormone.
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Etiologic factors in 1294 consecutive cases of male infertility.

TL;DR: Between January 1965 and December 1970 1294 cases of male subfertility were analyzed and it was found that artificial homologous insemination was often sucessful in each of these classes.
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Capacity of the Testosterone-Binding Globulin in Human Plasma and Influence of Specific Binding of Testosterone on Its Metabolic Clearance Rate

TL;DR: The binding capacity (TeBC) and binding index (BI) of the testosterone binding globulin (TeBG) have been studied in several groups of patients as discussed by the authors, including male hypogonadism in hyperthyroidism, and in males with chronic asthmatic bronchitis or cirrhosis of the liver.
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Studies on the Role of Sex Steroids in the Feedback Control of FSH Concentrations in Men

TL;DR: The data support the concept that although there is a specific seminiferous tubular factor regulating F SH secretion, testicular steroids also modulate FSH secretion.
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Secretion of Unconjugated Androgens and Estrogens by the Normal and Abnormal Human Testis before and after Human Chorionic Gonadotropin

TL;DR: E(1) is secreted by the human testis, but testicular secretion of E (1) accounts for less than 5% of E( 1) production in normal men, and strikingly decreased secretion of androgen and estrogen by unilateral atrophic human tests cannot be appreciated by analyses of peripheral steroid concentrations.
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