Journal ArticleDOI
Ultrastructural features of dopamine axon terminals in the anteromedial and the suprarhinal cortex of adult rat
TLDR
The ultrastructural features and synaptic relationships of dopamine (DA) axon terminals were examined in the prefrontal cortex of adult rat after immunocytochemical staining with a highly specific polyclonal antiserum directed against DA-glutaraldehyde-lysyl-protein conjugate.About:
This article is published in Brain Research.The article was published on 1988-02-23. It has received 156 citations till now. The article focuses on the topics: Cortex (anatomy) & Synaptic vesicle.read more
Citations
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Modulation of memory fields by dopamine D1 receptors in prefrontal cortex.
TL;DR: The precision shown for D1 receptor modulation of mnemonic processing indicates a direct gating of selective excitatory synaptic inputs to prefrontal neurons during cognition.
Book
Brainstem control of wakefulness and sleep
TL;DR: A monograph communicating the current realities and future possibilities of unifying basic studies on anatomy and cellular physiology with investigations of the behavioral and physiological events of waking and sleep.
Journal ArticleDOI
Prefrontal cortical efferents in the rat synapse on unlabeled neuronal targets of catecholamine terminals in the nucleus accumbens septi and on dopamine neurons in the ventral tegmental area.
TL;DR: The ultrastructural bases for these interactions in rat are investigated by examining the synaptic associations between prefrontal cortical terminals labeled with anterograde markers and neuronal processes containing immunoreactivity for the catecholamine synthesizing enzyme, tryosine hydroxylase.
Journal ArticleDOI
D1 receptors in prefrontal cells and circuits
TL;DR: These studies are based on the hypothesis that many features of schizophrenia represent a failure in the neural mechanisms by which prefrontal cortex stores and prowles information in working memory.
Journal ArticleDOI
Ventral Tegmental Area Neurons in Learned Appetitive Behavior and Positive Reinforcement
TL;DR: Identifying the neurotransmitter content and projection target of VTA neurons recorded in vivo will be critical for determining their contribution to learned appetitive behaviors.
References
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Journal Article
Evidence for the existence of monoamine-containing neurons in the central nervous system. i. demonstration of monoamines in the cell bodies of brain stem neurons.
Dahlstroem A,Fuxe K +1 more
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Dynamic response of chest wall and lung injuries in rabbits exposed to air shock waves of short duration.
Carl-Johan Clemedson,Joensson A +1 more
TL;DR: The chest walls of rabbits exposed to steep-fronted air shock waves of short duration produced by 50 g spherical charges of TNT were recorded by means of a mechano-electric motion transducer to study the correlation between the deformations of the chest and the damage inflicted to the underlying organs, especially the lungs.
Journal ArticleDOI
Distribution of serotonin-immunoreactivity in the central nervous system of the rat-cell bodies and terminals.
TL;DR: The results demonstrate a widespread occurrence of 5-HT-positive nerve terminals throughout the central nervous system and the finding that serotonin-containing neurons are present in many nuclei that also include catecholamine- containing neurons makes it necessary to reconsider the terminology of the monoaminergic cell groups.
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The projections of the ventral tegmental area and adjacent regions: A combined fluorescent retrograde tracer and immunofluorescence study in the rat
TL;DR: In the ventral part of the midbrain, essentially separate groups of aminergic and non-aminergic neurons in both the reticular formation (VTA) and in the adjacent nuclei of the raphe project bilaterally to a variety of similar terminal fields in the telencephalon, diencephal on, and brainstem.
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Cognitive deficit caused by regional depletion of dopamine in prefrontal cortex of rhesus monkey.
TL;DR: Evidence is provided that dopamine plays an important role in a specific cortical function in rhesus monkeys and that this role can be pharmacologically reversed with dopamine agonists.