A
Aldo Tagliabue
Researcher at National Research Council
Publications - 78
Citations - 3450
Aldo Tagliabue is an academic researcher from National Research Council. The author has contributed to research in topics: Immune system & Antigen. The author has an hindex of 32, co-authored 75 publications receiving 3306 citations. Previous affiliations of Aldo Tagliabue include Foundation for Biomedical Research & National Institutes of Health.
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Journal ArticleDOI
Multimer Formation and Ligand Recognition by the Long Pentraxin PTX3 SIMILARITIES AND DIFFERENCES WITH THE SHORT PENTRAXINS C-REACTIVE PROTEIN AND SERUM AMYLOID P COMPONENT
Barbara Bottazzi,Valérie Vouret-Craviari,Antonio Bastone,Luca De Gioia,Cristian Matteucci,Giuseppe Peri,Fabio Spreafico,M. Pausa,Cinzia D'Ettorre,Elisabetta Gianazza,Aldo Tagliabue,Mario Salmona,Francesco Tedesco,Martino Introna,Alberto Mantovani,Alberto Mantovani +15 more
TL;DR: The capacity to bind C1q, mediated by the pentraxin domain, is consistent with the view that PTX3, produced in tissues by endothelial cells or macrophages in response to interleukin-1 and tumor necrosis factor, may act as a local regulator of innate immunity.
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Characteristics of natural killer cells in the murine intestinal epithelium and lamina propria.
TL;DR: The phenotype of the gut NK cells appears to be different from the splenic one and provides further evidence for NK heterogeneity and establishes the compartmentalization of one NK subpopulation.
Journal ArticleDOI
The interleukin-1 receptor family.
Diana Boraschi,Aldo Tagliabue +1 more
TL;DR: The characteristics and functional roles of the IL-1R family are examined, which include the regulation of innate inflammation, host defense and acquired immunity.
Journal ArticleDOI
Changing Priorities in Vaccinology: Antibiotic Resistance Moving to the Top.
Aldo Tagliabue,Rino Rappuoli +1 more
TL;DR: This structure-based Ag design will bring to a new generation of vaccines able to target previously elusive infections, thereby offering an effective solution to the problem of AMR.
Journal Article
Interferon-gamma reduces macrophage-suppressive activity by inhibiting prostaglandin E2 release and inducing interleukin 1 production.
TL;DR: Normal peritoneal M phi of C3H/HeN mice were able to suppress lymphocyte proliferation in a dose-dependent fashion when added to Con A-pulsed spleen cell cultures, butM phi-suppressive activity could be partially or completely reduced by in vitro pre-exposure to nonimmune IFN-alpha or immune recombinantIFN-gamma.