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Alok C. Bharti

Researcher at University of Delhi

Publications -  102
Citations -  8483

Alok C. Bharti is an academic researcher from University of Delhi. The author has contributed to research in topics: Cancer & Cervical cancer. The author has an hindex of 38, co-authored 95 publications receiving 7744 citations. Previous affiliations of Alok C. Bharti include Indian Council of Medical Research & University of Texas MD Anderson Cancer Center.

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Therapeutic startegies for human papillomavirus infection and associated cancers.

TL;DR: An overview of the magnitude of the problem, current status of research on promising lead molecules, formulations and therapeutic strategies that showed potential to translate to clinically-viable HPV therapeutics to counteract this reproductive health challenge is provided.
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Higher prevalence of human papillomavirus infection in adolescent and young adult girls belonging to different Indian tribes with varied socio-sexual lifestyle.

TL;DR: This is a first study showing significantly a very high prevalence of HPV infection in adolescent and young adult tribal girls possibly due to different socio-sexual behavior, indicating a serious health concern for Indian tribal women.
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Expression of BRCA1 and BRCA2 proteins and their correlation with clinical staging in breast cancer.

TL;DR: A subset of cases that decline in BRCA1 expression that may be associated with potentially compensatory increase inBRCA2 protein, which may depend on tumor grade as well as menopausal status, are demonstrated.
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Anti-neoplastic action of aspirin against a T-cell lymphoma involves an alteration in the tumour microenvironment and regulation of tumour cell survival

TL;DR: A possible indirect involvement of the tumour microenvironment in addition to a direct but limited anti-neoplastic action of aspirin in the retardation of tumour growth is demonstrated.
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miRNA as viral transcription tuners in HPV-mediated cervical carcinogenesis.

TL;DR: A review of currently available miR data related to HPV infection and cervical carcinogenesis with a special focus on miRs that may regulate expression of the host and viral factors particularly responsible for viral transcription leading to carcinogenic progression of the lesion.