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Amina T. Tassa
Researcher at University of Texas Southwestern Medical Center
Publications - 4
Citations - 3487
Amina T. Tassa is an academic researcher from University of Texas Southwestern Medical Center. The author has contributed to research in topics: Autophagy & Autophagy database. The author has an hindex of 4, co-authored 4 publications receiving 3233 citations.
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Journal ArticleDOI
Bcl-2 antiapoptotic proteins inhibit Beclin 1-dependent autophagy.
Sophie Pattingre,Sophie Pattingre,Amina T. Tassa,Xueping Qu,Xueping Qu,Rita Garuti,Xiao Huan Liang,Noboru Mizushima,Milton Packer,Michael D. Schneider,Beth Levine,Beth Levine +11 more
TL;DR: Bcl-2 not only functions as an antiapoptotic protein, but also as an antiautophagy protein via its inhibitory interaction with Beclin 1, which may help maintain autophagy at levels that are compatible with cell survival, rather than cell death.
Journal ArticleDOI
Autophagy is required for G₁/G₀ quiescence in response to nitrogen starvation in Saccharomyces cerevisiae.
Z. H. An,Amina T. Tassa,Collin Thomas,Collin Thomas,Rui Zhong,Guanghua Xiao,Rati Fotedar,Benjamin P. Tu,Daniel J. Klionsky,Beth Levine +9 more
TL;DR: It is shown that autophagy genes regulate cell cycle arrest in the budding yeast Saccharomyces cerevisiae during nitrogen starvation, and this results suggest thatAutophagy is crucial for mitotic exit during starvation and appropriate entry into a G1/G0 quiescent state.
Journal ArticleDOI
Doxycycline Attenuates Isoproterenol- and Transverse Aortic Banding-Induced Cardiac Hypertrophy in Mice
Mounir Errami,Cristi L. Galindo,Amina T. Tassa,John M. DiMaio,Joseph A. Hill,Harold R. Garner +5 more
TL;DR: The results suggest that DOX might be evaluated as a potential CH therapeutic and also provide potential signaling mechanisms to investigate in the context of CH phenotype development and regression.
Journal ArticleDOI
Carbamazepine alone and in combination with doxycycline attenuates isoproterenol-induced cardiac hypertrophy.
Mounir Errami,Amina T. Tassa,Cristi L. Galindo,Michael A. Skinner,Joseph A. Hill,Harold R. Garner +5 more
TL;DR: Results suggest that carbamazepine acts as a β-adrenergic antagonist, and identifies 19 genes whose expression is significantly altered in treated animals and might be responsible for the added benefit provided by the combination therapy.