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Amitabha Mazumder

Researcher at New York University

Publications -  146
Citations -  10381

Amitabha Mazumder is an academic researcher from New York University. The author has contributed to research in topics: Multiple myeloma & Bortezomib. The author has an hindex of 40, co-authored 146 publications receiving 9842 citations. Previous affiliations of Amitabha Mazumder include New York Medical College & Westchester Medical Center.

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Lymphokine-activated killer cell phenomenon. Lysis of natural killer-resistant fresh solid tumor cells by interleukin 2-activated autologous human peripheral blood lymphocytes.

TL;DR: Evidence is presented that the LAK system is a phenomenon distinct from either NK or CTL systems that probably accounts for a large number of reported nonclassical cytotoxicities and may be functional in immune surveillance against human solid tumors.
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Daratumumab monotherapy in patients with treatment-refractory multiple myeloma (SIRIUS): an open-label, randomised, phase 2 trial

TL;DR: Daratumumab monotherapy showed encouraging efficacy in heavily pretreated and refractory patients with multiple myeloma, with a favourable safety profile in this population of patients.
Journal Article

Lysis of Fresh and Cultured Autologous Tumor by Human Lymphocytes Cultured in T-Cell Growth Factor

TL;DR: It is demonstrated that peripheral lymphoid cells grown in TCGF can be lytic for autologous cultured and autOLOGous fresh tumor compared to the lysis of fresh autologOUS peripheral blood lymphocytes.
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Lymphokine-activated killer cell phenomenon. II. Precursor phenotype is serologically distinct from peripheral T lymphocytes, memory cytotoxic thymus-derived lymphocytes, and natural killer cells

TL;DR: While the cell(s) responsible for activation and expression of LAK activity have some common features with the classic T cell-mediated CTL and NK cell systems, the LAK precursor cells are clearly distinct as determined by phenotype analysis using monoclonal antibodies and complement, and at present must be classified as a “null” cell.