scispace - formally typeset
A

Anna Paini

Researcher at University of Brescia

Publications -  112
Citations -  4111

Anna Paini is an academic researcher from University of Brescia. The author has contributed to research in topics: Blood pressure & Left ventricular hypertrophy. The author has an hindex of 23, co-authored 94 publications receiving 3426 citations. Previous affiliations of Anna Paini include Paris Descartes University.

Papers
More filters
Journal ArticleDOI

Determinants of pulse wave velocity in healthy people and in the presence of cardiovascular risk factors: 'establishing normal and reference values'.

TL;DR: In this paper, the authors established reference and normal values for Carotid-femoral pulse wave velocity (PWV), a direct measure of aortic stiffness, based on a large European population.
Journal ArticleDOI

Left Ventricular Concentric Geometry During Treatment Adversely Affects Cardiovascular Prognosis in Hypertensive Patients

TL;DR: Left ventricular (LV) mass and geometry predict risk for cardiovascular events in hypertension and regression of LV hypertrophy (LVH) may imply an important prognostic significance in hypertensive patients with and without LVH.
Journal ArticleDOI

Carotid and Aortic Stiffness: Determinants of Discrepancies

TL;DR: AlthoughCarotid-femoral pulse wave velocity and carotid stiffness provided similar information on the impact of aging on large artery stiffness in normal subjects, this was not the case for high blood pressure and/or diabetes.
Journal ArticleDOI

Ethnic-Specific Normative Reference Values for Echocardiographic LA and LV Size, LV Mass, and Systolic Function: The EchoNoRMAL Study

Katrina Poppe, +124 more
TL;DR: Sex- and/or ethnic-appropriate echocardiographic reference values are indicated for many measurements of LA and LV size, LV mass, and EF, and Reference values for LV volumes and mass also differ across the age range.
Journal Article

Inappropriate Left Ventricular Mass in Patients With Primary Aldosteronism. Commentary

TL;DR: In patients with PA, the prevalence of inappropriate LVM is increased, even in the absence of traditionally defined left ventricular hypertrophy, and the increase in aldosterone levels could contribute to the increase of LV mass exceeding the amount needed to compensate hemodynamic load.