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Anne Sofie Andreasen

Researcher at University of Copenhagen

Publications -  29
Citations -  3518

Anne Sofie Andreasen is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Medicine & Intensive care unit. The author has an hindex of 10, co-authored 20 publications receiving 2758 citations. Previous affiliations of Anne Sofie Andreasen include Copenhagen University Hospital & Herlev Hospital.

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Gut Microbiota in Human Adults with Type 2 Diabetes Differs from Non-Diabetic Adults

TL;DR: The results of this study indicate that type 2 diabetes in humans is associated with compositional changes in intestinal microbiota and the level of glucose tolerance should be considered when linking microbiota with metabolic diseases such as obesity and developing strategies to control metabolic diseases by modifying the gut microbiota.
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Effects of Lactobacillus acidophilus NCFM on insulin sensitivity and the systemic inflammatory response in human subjects

TL;DR: Insulin sensitivity was preserved among volunteers in the L. acidophilus NCFM group, whereas it decreased in the placebo group, and the systemic inflammatory response were, however, unaffected by the intervention.
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Human Endotoxemia as a Model of Systemic Inflammation

TL;DR: The activation of inflammatory cascades as well as organ-specific haemodynamic and functional changes after lipopolysaccharide are described, and the limitations of human-experimental models for the study of clinical disease are discussed.
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Lower or Higher Oxygenation Targets for Acute Hypoxemic Respiratory Failure

TL;DR: In this paper, the benefits and harms of different oxygenation levels for patients with acute hypoxemic respiratory failure in the intensive care unit (ICU) were discussed. But, the benefits of different levels of oxygenation were not evaluated.
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Type 2 diabetes is associated with altered NF-κB DNA binding activity, JNK phosphorylation, and AMPK phosphorylation in skeletal muscle after LPS.

TL;DR: It is indicated that pathways regulating glucose uptake in skeletal muscle may be involved in the development of inflammation-associated hyperglycemia and patients with type 2 diabetes exhibit changes in these pathways, which may ultimately render such patients more prone to develop dysregulated glucose disposal in the context of systemic inflammation.