A
Arthur M. Feldman
Researcher at Thomas Jefferson University
Publications - 296
Citations - 41891
Arthur M. Feldman is an academic researcher from Thomas Jefferson University. The author has contributed to research in topics: Heart failure & Tumor necrosis factor alpha. The author has an hindex of 79, co-authored 289 publications receiving 40355 citations. Previous affiliations of Arthur M. Feldman include Johns Hopkins University School of Medicine & Valley Hospital.
Papers
More filters
Journal ArticleDOI
TRPM2 Channels Protect against Cardiac Ischemia-Reperfusion Injury ROLE OF MITOCHONDRIA
Barbara A. Miller,Nicholas E. Hoffman,Salim Merali,Xue-Qian Zhang,JuFang Wang,Sudarsan Rajan,Santhanam Shanmughapriya,Erhe Gao,Carlos A. Barrero,Karthik Mallilankaraman,Jianliang Song,Tongda Gu,Iwona Hirschler-Laszkiewicz,Walter J. Koch,Arthur M. Feldman,Muniswamy Madesh,Joseph Y. Cheung +16 more
TL;DR: It is concluded that TRPM2 channels protect the heart from I/R injury by ameliorating mitochondrial dysfunction and reducing reactive oxygen species levels.
Journal ArticleDOI
Enhanced External Counterpulsation in Patients With Heart Failure: A Multicenter Feasibility Study
Ozlem Soran,Bruce Fleishman,Theresa Demarco,William Grossman,Virginia Schneider,Karen Manzo,Paul-Andre de Lame,Arthur M. Feldman +7 more
TL;DR: It is suggested that enhanced externalcounterpulsation is safe and well tolerated in patients with stable heart failure, and that a randomized, controlled study of enhanced external counterpulsations in these patients is warranted.
Journal ArticleDOI
Cerebrospinal fluid production: stimulation by cholera toxin.
TL;DR: Large increases in the production of cerebrospinal fluid have been observed after the intraventricular administration of cholera toxin, and the data suggest that adenosine 3',5'-monophosphate plays a role in cerebroSpinal fluid production.
Journal ArticleDOI
Endomyocardial gene expression during development of pacing tachycardia-induced heart failure in the dog.
R E Williams,David A. Kass,Y Kawagoe,P Pak,Richard S. Tunin,R Shah,A Hwang,Arthur M. Feldman +7 more
TL;DR: Levels of mRNA encoding beta-myosin heavy chain, mitochondrial creatine kinase, phospholamban, and sarcoplasmic reticulum Ca(2+)-ATPase did not significantly change from baseline, despite development of heart failure.
Journal ArticleDOI
Mitochondrial Abnormalities in Tumor Necrosis Factor-α–Induced Heart Failure Are Associated With Impaired DNA Repair Activity
TL;DR: Cardiac mtDNA repair mechanisms, mtDNA damage, and mitochondrial structure and function in mice with heart failure secondary to overexpression of TNF-&agr; (TNF1.6 mice) are studied to suggest that mtDNA mutations and mitochondrial structural and functional alterations in TNF–induced heart failure may be associated with reduced mt DNA repair activity.