A
Arthur M. Feldman
Researcher at Thomas Jefferson University
Publications - 296
Citations - 41891
Arthur M. Feldman is an academic researcher from Thomas Jefferson University. The author has contributed to research in topics: Heart failure & Tumor necrosis factor alpha. The author has an hindex of 79, co-authored 289 publications receiving 40355 citations. Previous affiliations of Arthur M. Feldman include Johns Hopkins University School of Medicine & Valley Hospital.
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Journal ArticleDOI
Up-regulation of beta 2-adrenergic receptors in previously transplanted, denervated nonfailing human hearts.
Hanadi M. Farrukh,Michel White,J. D. Port,David Handwerger,Patti Larrabee,Jana Klein,Robert Roden,Lisa Skerl,Dale G. Renlund,Arthur M. Feldman,Michael R. Bristow +10 more
TL;DR: These data indicate that the transplanted human heart exhibits an up-regulation of functional beta 2-adrenergic receptors, similar in the transplant and donor groups.
Journal ArticleDOI
Rationale and design of the treatment of hyponatremia based on lixivaptan in NYHA class III/IV cardiac patient evaluation (THE BALANCE) study.
William T. Abraham,Juan M. Aranda,John P. Boehmer,Uri Elkayam,Edward M. Gilbert,Stephen S. Gottlieb,Gerd Hasenfuβ,Marrick Kukin,Brian D. Lowes,John B. O’Connell,Luigi Tavazzi,Arthur M. Feldman,Barry Ticho,Cesare Orlandi +13 more
TL;DR: Lixivaptan was shown to increase serum sodium and reduce body weight, without renal dysfunction or hypokalemia, and improve hyponatremia by promoting electrolyte‐free aquaresis without significant side effects.
Journal ArticleDOI
Does academic culture support translational research
TL;DR: Academic medical centers must build new structures and modify their cultures to facilitate the development of seamless collaboration and cooperation among diverse groups of investigators.
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Phospholemman and β-adrenergic stimulation in the heart
JuFang Wang,Erhe Gao,Jianliang Song,Xue-Qian Zhang,Jifen Li,Walter J. Koch,Amy L. Tucker,Kenneth D. Philipson,Tung O. Chan,Arthur M. Feldman,Joseph Y. Cheung +10 more
TL;DR: Under stressful conditions in which [Na(+)](i) was high, beta-adrenergic agonist-mediated phosphorylation of PLM resulted in time-dependent reduction in inotropy due to relief of inhibition of Na(+)-K(+-ATPase.
Journal ArticleDOI
The human phospholamban gene: structure and expression.
Charles F. McTiernan,Carole S. Frye,Bonnie Lemster,Eric A. Kinder,Monique L. Ogletree-Hughes,Christine S. Moravec,Arthur M. Feldman +6 more
TL;DR: Transient transfection assays with a series of plasmids containing deleted 5' flanking regions showed that sequences between -169 and the CP1-box at -93 were required for maximal promoter activity in neonatal rat cardiomyocytes, suggesting at least a partial tissue selectivity of these reporter constructs.