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Barry J. Dickson

Researcher at Howard Hughes Medical Institute

Publications -  147
Citations -  24169

Barry J. Dickson is an academic researcher from Howard Hughes Medical Institute. The author has contributed to research in topics: fruitless & Axon guidance. The author has an hindex of 70, co-authored 139 publications receiving 22099 citations. Previous affiliations of Barry J. Dickson include University of Queensland & Research Institute of Molecular Pathology.

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A genome-wide transgenic RNAi library for conditional gene inactivation in Drosophila

TL;DR: The generation and validation of a genome-wide library of Drosophila melanogaster RNAi transgenes, enabling the conditional inactivation of gene function in specific tissues of the intact organism and opening up the prospect of systematically analysing gene functions in any tissue and at any stage of the Drosophile lifespan.
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Molecular Mechanisms of Axon Guidance

TL;DR: This work has shown that a relatively small number of guidance factors can be used to generate intricate patterns of neuronal wiring through signaling pathways still only poorly understood.
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Molecular, anatomical, and functional organization of the Drosophila olfactory system

TL;DR: These Or expression and ORN connectivity maps provide further insight into the molecular, anatomical, and functional organization of the Drosophila olfactory system and provide an essential resource for investigating how internal odor representations are generated and how they are further processed and transmitted to higher brain centers.
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A single class of olfactory neurons mediates behavioural responses to a Drosophila sex pheromone.

TL;DR: It is shown that the Drosophila melanogaster male-specific pheromone 11-cis-vaccenyl acetate (cVA) acts through the receptor Or67d to regulate both male and female mating behaviour, and it is suggested that cVA has opposite effects in the two sexes: inhibiting mating behaviour in males but promoting mating behaviours in females.
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Analysis of Drosophila photoreceptor axon guidance in eye-specific mosaics

TL;DR: It is suggested that PTP69D plays a permissive role, perhaps reducing the adhesion of R1-R6 and R7 growth cones to the pioneer R8 axon so that they can respond independently to their specific targeting cues.