B
Betty Soliven
Researcher at University of Chicago
Publications - 90
Citations - 4407
Betty Soliven is an academic researcher from University of Chicago. The author has contributed to research in topics: Myasthenia gravis & Myelin. The author has an hindex of 36, co-authored 85 publications receiving 4026 citations. Previous affiliations of Betty Soliven include NewYork–Presbyterian Hospital & University of Illinois at Chicago.
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Journal ArticleDOI
Fingolimod (FTY720) enhances remyelination following demyelination of organotypic cerebellar slices.
Veronique E. Miron,Samuel K. Ludwin,Peter J. Darlington,Andrew A. Jarjour,Andrew A. Jarjour,Betty Soliven,Timothy E. Kennedy,Jack P. Antel +7 more
TL;DR: Data demonstrate that fingolimod modulates multiple neuroglial cell responses, resulting in enhanced remyelination in organotypic slice cultures that maintain the complex cellular interactions of the mammalian brain.
Journal ArticleDOI
FTY720 modulates human oligodendrocyte progenitor process extension and survival.
Veronique E. Miron,Cha-Gyun Jung,Hye-Jung Kim,Timothy E. Kennedy,Betty Soliven,Jack P. Antel +5 more
TL;DR: The objective was to assess the effect of FTY720 on process extension, differentiation, and survival of human oligodendrocyte progenitor cells (OPCs), and link the functional effects with S1P receptor expression and signaling.
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Association of TAG-1 with Caspr2 is essential for the molecular organization of juxtaparanodal regions of myelinated fibers
Maria Traka,Laurence Goutebroze,Natalia Denisenko,Maria Bessa,Artemisia Nifli,Sophia Havaki,Yoichiro Iwakura,Fumihiko Fukamauchi,Kazutada Watanabe,Betty Soliven,Jean-Antoine Girault,Domna Karagogeos +11 more
TL;DR: A tripartite intercellular protein complex, comprised of these two proteins, appears critical for axo-glial contacts at juxtaparanodes, suggesting that similar molecules are crucial for different types of axo–glial interactions.
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Development of Spontaneous Autoimmune Peripheral Polyneuropathy in B7-2–Deficient Nod Mice
Benoît L. Salomon,Lesley Rhee,Hélène Bour-Jordan,Hélène Bour-Jordan,Honor Hsin,Anthony G. Montag,Betty Soliven,Jennifer Arcella,Ann M. Girvin,Stephen D. Miller,Jeffrey A. Bluestone +10 more
TL;DR: The B7-2–deficient NOD mouse constitutes the first model of a spontaneous autoimmune disease of the peripheral nervous system, which has many similarities to the human disease, chronic inflammatory demyelinating polyneuropathy (CIDP).
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Proprioceptive Sensory Neuropathy in Mice with a Mutation in the Cytoplasmic Dynein Heavy Chain 1 Gene
Xiang Jun Chen,Eleni N. Levedakou,Kathleen J. Millen,Robert L. Wollmann,Betty Soliven,Brian Popko +5 more
TL;DR: In vivo evidence is provided that distinct mutations in cytoplasmic dynein can either result in a pure sensory neuropathy or in a sensory Neuropathy with motor neuron involvement.