Bianca Maria Scicchitano

TL;DR: A novel in vivo role for AVP-dependent pathways is highlighted, which may represent an interesting strategy to counteract muscle decline in aging or in muscular pathologies.

TL;DR: The aim of this review is to recapitulate the pathological features of GBM and of the peritumoral region associated with progression and recurrence and to suggest new immunotherapeutic strategies.

TL;DR: It is reported that only IGF‐1Ea expression promotes a pronounced hypertrophic phenotype in young mice, which is maintained in aged mice, and data suggest that IGF‐ 1 is a promising therapeutic agent in staving off advancing muscle weakness.

TL;DR: It is shown that patterns ultimately emanate from dedicated pigment-receiving cells, and that recipients provide a cutaneous template or blueprint that instructs melanocytes where to place pigment.

TL;DR: This study demonstrated that although the regenerative program can be impaired by the limited proliferative capacity of satellite cells, this limit is not reached during normal aging, and it is more likely that the restricted muscle repair program in aging is presumably due to missing signals that usually render the damaged muscle a permissive environment for regenerative activity.