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Celeste Allaband

Researcher at University of California, San Diego

Publications -  13
Citations -  403

Celeste Allaband is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Microbiome & Biology. The author has an hindex of 5, co-authored 9 publications receiving 189 citations. Previous affiliations of Celeste Allaband include Colorado State University.

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Microbiome 101: Studying, Analyzing, and Interpreting Gut Microbiome Data for Clinicians.

TL;DR: The content of the human microbiome, including intersubject and intrasubject variability, considerations of study design including important confounding factors, and different methods in the laboratory and on the computer to read the microbiome and its resulting gene products and metabolites are discussed.
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Human Skin, Oral, and Gut Microbiomes Predict Chronological Age.

TL;DR: The skin microbiome provides the best prediction of age, suggesting a model in which physiological aging occurs concomitantly with the loss of key taxa over a lifetime, enabling potential microbiome-targeted therapeutic strategies to prevent aging.
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Engineering the microbiome for animal health and conservation

TL;DR: The rapid progress currently being made in human medicine and the examples to date of application of probiotics and other microbiome-directed therapies in taxa ranging from horses to salamanders to bees suggest excellent prospects for these technologies as they are further developed.
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Wildlife-microbiome interactions and disease: exploring opportunities for disease mitigation across ecological scales

TL;DR: Investigation of wildlife affords a unique opportunity to gain understanding of the broad diversity of the associated microbiota and learn from nature’s molecular and microbial responses to disease.
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Intermittent Hypoxia and Hypercapnia Alter Diurnal Rhythms of Luminal Gut Microbiome and Metabolome.

TL;DR: In this article, the authors found that IHC significantly changes the diurnal dynamics of the colon microbiome and metabolome, increasing members and metabolites that are proinflammatory and proatherogenic while decreasing protective ones.