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Cynthia J. Meininger

Researcher at Texas A&M University

Publications -  123
Citations -  11763

Cynthia J. Meininger is an academic researcher from Texas A&M University. The author has contributed to research in topics: Arginine & Nitric oxide. The author has an hindex of 53, co-authored 122 publications receiving 10819 citations. Previous affiliations of Cynthia J. Meininger include Oklahoma State University–Stillwater & Veterans Health Administration.

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Deletion of Cdc42 Enhances ADAM17-Mediated Vascular Endothelial Growth Factor Receptor 2 Shedding and Impairs Vascular Endothelial Cell Survival and Vasculogenesis

TL;DR: The inactivation of Cdc42 in ECs decreased the level of vascular endothelial growth factor receptor 2 (VEGFR2) protein on the EC surface and promoted the production of a 75-kDa membrane-associated C-terminal VEGFR2 fragment, indicating that CDC42 is essential for VEGfr2-mediated signal transduction in blood vessel formation.
Journal Article

l-Glutamine Inhibits Nitric Oxide Synthesis in Bovine Venular Endothelial Cells

TL;DR: Regulation of the arginine-NO pathway by L-glutamine may have pharmacologic and therapeutic implications in such conditions as inflammation and septic shock by inhibiting NO generation from L-arginine in endothelial cells.
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Adverse organogenesis and predisposed long-term metabolic syndrome from prenatal exposure to fine particulate matter.

TL;DR: Maternal exposure to ultrafine ammonium sulfate aerosols impacts prenatal and postnatal organogenesis in offspring and predisposes metabolic syndrome in adult life and highlights the necessity to develop therapeutic strategies to remedy adverse health effects of maternal PM exposure on conceptus/postnatal growth and development.
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Effects of 28 days of beta-alanine and creatine supplementation on muscle carnosine, body composition and exercise performance in recreationally active females

TL;DR: Although mean changes were consistent with prior studies and large effect sizes were noted, no significant differences were observed among groups in changes in muscle carnosine levels and the impact of CRE supplementation appeared to diminish during the maintenance phase.