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David A. Kass
Researcher at Johns Hopkins University School of Medicine
Publications - 605
Citations - 63963
David A. Kass is an academic researcher from Johns Hopkins University School of Medicine. The author has contributed to research in topics: Heart failure & Cardiac resynchronization therapy. The author has an hindex of 127, co-authored 580 publications receiving 58747 citations. Previous affiliations of David A. Kass include University of Pittsburgh & Johns Hopkins University.
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Journal ArticleDOI
L-arginine therapy in acute myocardial infarction: the Vascular Interaction With Age in Myocardial Infarction (VINTAGE MI) randomized clinical trial.
Steven P. Schulman,Lewis C. Becker,David A. Kass,Hunter C. Champion,Michael L. Terrin,Sandra A. Forman,Kavita V Ernst,Kavita V Ernst,Mark D. Kelemen,Mark D. Kelemen,Susan N. Townsend,Anne Capriotti,Joshua M. Hare,Gary Gerstenblith +13 more
TL;DR: There was no significant change from baseline to 6 months in the vascular stiffness measurements or left ventricular ejection fraction in either of the 2 groups, including those 60 years or older and the entire study group.
Journal ArticleDOI
From Emax to pressure-volume relations: a broader view
David A. Kass,W L Maughan +1 more
TL;DR: This approach was originally studied as a means of integrating and modeling the heart and vascular systems to assess "the effects of different forms of heart disease and to predict responses to the many therapeutic options now available for the treatment of the patient with cardiac disease."
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Mechanisms Underlying Conduction Slowing and Arrhythmogenesis in Nonischemic Dilated Cardiomyopathy
TL;DR: HF was associated with marked prolongation of the QRS duration of the volume conducted electrocardiogram and significant slowing of epicardial and endocardial conduction velocities (CV) in both LV and RV and changes in phosphorylation and localization of Cx43 may contribute to gap-junction dysfunction, CV slowing, and arrhythmias in HF.
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Nitroxyl anion exerts redox-sensitive positive cardiac inotropy in vivo by calcitonin gene-related peptide signaling.
Nazareno Paolocci,Walter F. Saavedra,Katrina M. Miranda,Cristian Martignani,Takayoshi Isoda,Joshua M. Hare,Michael Graham Espey,Jon M. Fukuto,Martin Feelisch,David A. Wink,David A. Kass +10 more
TL;DR: No− is a redox-sensitive positive inotrope with selective venodilator action, whose cardiac effects are mediated by CGRP-receptor stimulation, and may prove useful for the treatment of cardiovascular diseases characterized by cardiac depression and elevated venous filling pressures.
Journal ArticleDOI
Impact of Arterial Load and Loading Sequence on Left Ventricular Tissue Velocities in Humans
Barry A. Borlaug,Vojtech Melenovsky,Margaret M. Redfield,Kristy Kessler,Hyuk Jae Chang,Theodore P. Abraham,David A. Kass +6 more
TL;DR: Diastolic and systolic tissue velocities vary inversely with arterial afterload, with late-systolic load having the greatest influence on E'.