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David H. Munn

Researcher at Georgia Regents University

Publications -  255
Citations -  35440

David H. Munn is an academic researcher from Georgia Regents University. The author has contributed to research in topics: Immune system & T cell. The author has an hindex of 83, co-authored 246 publications receiving 31855 citations. Previous affiliations of David H. Munn include Hokkaido University & University of Nebraska Medical Center.

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Prevention of allogeneic fetal rejection by tryptophan catabolism

TL;DR: In 1953 Medawar pointed out that survival of the genetically disparate (allogeneic) mammalian conceptus contradicts the laws of tissue transplantation and suppresses T cell activity and defends itself against rejection.
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IDO expression by dendritic cells: tolerance and tryptophan catabolism.

TL;DR: This review summarizes key recent developments and proposes a unifying model for the role of IDO in tolerance induction, including studies of mammalian pregnancy, tumour resistance, chronic infections and autoimmune diseases.
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Activation of Gpr109a, Receptor for Niacin and the Commensal Metabolite Butyrate, Suppresses Colonic Inflammation and Carcinogenesis

TL;DR: This paper showed that GPR109a signaling promoted anti-inflammatory properties in colonic macrophages and dendritic cells and enabled them to induce differentiation of Treg cells and IL-10-producing T cells.
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Inhibition of t cell proliferation by macrophage tryptophan catabolism

TL;DR: It is shown that monocytes that have differentiated under the influence of macrophage colony-stimulating factor acquire the ability to suppress T cell proliferation in vitro via rapid and selective degradation of tryptophan by IDO.
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GCN2 kinase in T cells mediates proliferative arrest and anergy induction in response to indoleamine 2,3-dioxygenase.

TL;DR: It is shown that IDO-expressing plasmacytoid DCs activate the GCN2 kinase pathway in responding T cells, allowing them to detect and respond to conditions created by IDO.