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David Sanchez-Migallon Guzman

Researcher at University of California, Davis

Publications -  224
Citations -  6920

David Sanchez-Migallon Guzman is an academic researcher from University of California, Davis. The author has contributed to research in topics: Population & Amazona ventralis. The author has an hindex of 36, co-authored 201 publications receiving 5848 citations. Previous affiliations of David Sanchez-Migallon Guzman include Royal Free Hospital & San Francisco General Hospital.

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Clinical picture and treatment of 2212 patients with common variable immunodeficiency

TL;DR: In this article, the authors analyzed the clinical presentation, association between clinical features, and differences and effects of immunoglobulin treatment in Europe for Common Variable Immunodeficiency (CVID) patients.
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Measuring adherence to antiretroviral therapy in a diverse population using a visual analogue scale.

TL;DR: A visual analogue scale to assess adherence was performed as well as a more complicated 3-day recall instrument in this diverse population of HIV-infected patients, and may be useful in routine patient care.
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Adherence-resistance relationships for protease and non-nucleoside reverse transcriptase inhibitors explained by virological fitness.

TL;DR: NNRTI resistance was more common than PI resistance at low levels of adherence, and class-specific adherence–resistance relationships are associated with the relative replicative capacity of drug-resistant versus wild-type variants to replicate in the presence of clinically relevant drug levels.
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Cost-effectiveness of PCSK9 Inhibitor Therapy in Patients With Heterozygous Familial Hypercholesterolemia or Atherosclerotic Cardiovascular Disease

TL;DR: Using PCSK9 inhibitor use in patients with heterozygous FH or ASCVD did not meet generally acceptable incremental cost-effectiveness thresholds and was estimated to increase US health care costs substantially, compared with initiating statins in all statin-tolerant individuals who are not currently using statins.
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High levels of adherence do not prevent accumulation of HIV drug resistance mutations.

TL;DR: Increasing rates of viral suppression at high levels of adherence is balanced by increasing rates of drug resistance among viremic patients, which will not prevent population levels ofdrug resistance.