D
Dina Ragab
Researcher at Ain Shams University
Publications - 23
Citations - 1261
Dina Ragab is an academic researcher from Ain Shams University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 7, co-authored 15 publications receiving 592 citations. Previous affiliations of Dina Ragab include Pasteur Institute.
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Journal ArticleDOI
The COVID-19 Cytokine Storm; What We Know So Far.
TL;DR: Targeting cytokines during the management of COVID-19 patients could improve survival rates and reduce mortality.
Journal ArticleDOI
Longer duration of kangaroo care improves neurobehavioral performance and feeding in preterm infants: a randomized controlled trial.
Rania A. El-Farrash,Dina M. Shinkar,Dina Ragab,Ramy M. Salem,Wessam E Saad,Ahmed S Farag,Dina H Salama,Medhat F Sakr +7 more
TL;DR: Preterm neonates who receive kangaroo care for long durations reach full enteral feeds faster, have better breastfeeding success, neurobehavioral performance, thermal control, and tissue oxygenation.
Journal ArticleDOI
CXCL10 antagonism and plasma sDPPIV correlate with increasing liver disease in chronic HCV genotype 4 infected patients
Dina Ragab,Melissa E. Laird,Darragh Duffy,Darragh Duffy,Armanda Casrouge,Armanda Casrouge,Rasha Mamdouh,Amal Abass,Dina El. Shenawy,Abdelhadi M. Shebl,Wagdi Elkashef,Khaled Zalata,Mostafa Kamal,Gamal Esmat,Philippe Bonnard,Arnaud Fontanet,Arnaud Fontanet,Mona Rafik,Matthew L. Albert,Matthew L. Albert,Matthew L. Albert +20 more
TL;DR: The observation of chemokine antagonism as a mechanism of immune modulation in chronic HCV patients is generalized and may help guide the use of new therapeutic immune modulators.
Journal ArticleDOI
Vitamin D status and its modulatory effect on interferon gamma and interleukin-10 production by peripheral blood mononuclear cells in culture
TL;DR: This study demonstrates that vitamin D modulates IFN-γ and IL-10 production and provides a rationale for evaluating vitamin D as an immunomodulatory agent.
Journal ArticleDOI
Elevated levels of IL-37 correlate with T cell activation status in rheumatoid arthritis patients.
TL;DR: The results suggest that in an active disease status, activated T lymphocytes may be a contributing source to the elevated levels of IL‐37 trying to down‐regulate the active inflammatory process.