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E. Kale Edmiston

Researcher at University of Pittsburgh

Publications -  30
Citations -  831

E. Kale Edmiston is an academic researcher from University of Pittsburgh. The author has contributed to research in topics: Transgender & Autism. The author has an hindex of 11, co-authored 23 publications receiving 377 citations. Previous affiliations of E. Kale Edmiston include Allen Institute for Brain Science & Vanderbilt University.

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Journal ArticleDOI

Standards of Care for the Health of Transgender and Gender Diverse People, Version 8

Eli Coleman, +118 more
TL;DR: The SOC-8 guidelines are intended to be flexible to meet the diverse health care needs of TGD people globally and offer standards for promoting optimal health care and guidance for the treatment of people experiencing gender incongruence.
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Opportunities and Gaps in Primary Care Preventative Health Services for Transgender Patients: A Systemic Review.

TL;DR: Transgender people face barriers to accessing healthcare, resulting in population-level disparities in health outcomes, and little research is available to better understand the receipt of primary healthcare among transgender patients or how the rate of receipt of preventive care may differ among transgender populations.
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Neurobiological Commonalities and Distinctions Among Three Major Psychiatric Diagnostic Categories: A Structural MRI Study

TL;DR: The findings of common alterations in SZ, BD, and MDD support the presence of core neurobiological disruptions in these disorders and suggest that neural structural distinctions between these disorders may be less prominent than initially postulated, particularly between SZ and BD.
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Altered resting-state functional connectivity of the dentate nucleus in Parkinson's disease.

TL;DR: It is suggested that connectivity of the DN in the resting state is disrupted in Parkinson's disease, and there may be a compensatory cerebellar connectivity mechanism in the rested state in PD.
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Structure-function associations in hippocampus in bipolar disorder.

TL;DR: Associations between morphological changes in hippocampus structure in BD and verbal memory impairment are supported and provide preliminary evidence pharmacotherapy may reverse hippocampus-related memory deficits.