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Eduardo Soriano

Researcher at University of Barcelona

Publications -  233
Citations -  16500

Eduardo Soriano is an academic researcher from University of Barcelona. The author has contributed to research in topics: Hippocampal formation & Cerebral cortex. The author has an hindex of 72, co-authored 225 publications receiving 15459 citations. Previous affiliations of Eduardo Soriano include French Institute of Health and Medical Research & Catalan Institution for Research and Advanced Studies.

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Regional and cellular patterns of reelin mRNA expression in the forebrain of the developing and adult mouse

TL;DR: High-resolution in situ hybridization analyses to determine the pattern of reelin expression in the developing forebrain of the mouse revealed a complex pattern of cellular and regional expression that is consistent with Reelin having multiple roles in brain development and adult brain function.
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A role for Cajal–Retzius cells and reelin in the development of hippocampal connections

TL;DR: It is reported that ablation of Cajal–Retzius cells in organotypic slice cultures of hippocampus prevented the ingrowth of entorhinal but not of commissural afferents, and reelin is essential for the formation of layer-specific hippocampal connections.
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Mechanism suppressing glycogen synthesis in neurons and its demise in progressive myoclonus epilepsy.

TL;DR: It is shown that mouse neurons have the enzymatic machinery for synthesizing glycogen, but that it is suppressed by retention of muscle glycogen synthase in the phosphorylated, inactive state, and that the malin-laforin complex ensures a blockade of neuronal glycogen synthesis even under intense glycogenic conditions.
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BDNF regulates spontaneous correlated activity at early developmental stages by increasing synaptogenesis and expression of the K+/Cl- co-transporter KCC2.

TL;DR: Using transgenic embryos that overexpress BDNF under the control of the nestin promoter, it is shown that BDNF controls the emergence and robustness of spontaneous activity in embryonic hippocampal slices and dramatically increases spontaneous co-active network activity.