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Estelle Bettelli

Researcher at Harvard University

Publications -  15
Citations -  13608

Estelle Bettelli is an academic researcher from Harvard University. The author has contributed to research in topics: Experimental autoimmune encephalomyelitis & Myelin oligodendrocyte glycoprotein. The author has an hindex of 9, co-authored 15 publications receiving 12669 citations. Previous affiliations of Estelle Bettelli include Brigham and Women's Hospital.

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Journal ArticleDOI

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells.

TL;DR: It is shown that IL-6, an acute phase protein induced during inflammation, completely inhibits the generation of Foxp3+ Treg cells induced by TGF-β, and the data demonstrate a dichotomy in thegeneration of pathogenic (TH17) T cells that induce autoimmunity and regulatory (Foxp3+) T Cells that inhibit autoimmune tissue injury.
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IL-17 and Th17 Cells.

TL;DR: The investigation of the differentiation, effector function, and regulation of Th17 cells has opened up a new framework for understanding T cell differentiation and now appreciate the importance of Th 17 cells in clearing pathogens during host defense reactions and in inducing tissue inflammation in autoimmune disease.
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Loss of T-bet, But Not STAT1, Prevents the Development of Experimental Autoimmune Encephalomyelitis

TL;DR: Enhanced EAE in STAT1−/− mice was associated with continued generation of IFN-γ–producing Th1 cells and up-regulation of selective chemokines responsible for the increased recruitment of macrophages and neutrophils in the central nervous system.
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IL-9 induces differentiation of TH17 cells and enhances function of FoxP3+ natural regulatory T cells

TL;DR: It is demonstrated that IL-9 is a key molecule that affects differentiation of TH17 cells and Treg function, and a role of IL- 9 as a regulator of pathogenic versus protective mechanisms of immune responses is highlighted.