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Gereon R. Fink

Researcher at Forschungszentrum Jülich

Publications -  976
Citations -  67974

Gereon R. Fink is an academic researcher from Forschungszentrum Jülich. The author has contributed to research in topics: Medicine & Functional magnetic resonance imaging. The author has an hindex of 114, co-authored 867 publications receiving 60853 citations. Previous affiliations of Gereon R. Fink include University of Geneva & University of Hamburg.

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Dose-dependent effects of theta burst rTMS on cortical excitability and resting-state connectivity of the human motor system.

TL;DR: Investigating whether increasing the number of pulses of intermittent TBS (iTBS) increases cortical excitability as measured by motor-evoked potentials (MEPs) and alters functional connectivity measured using resting-state fMRI, in a dose-dependent manner results in dose- dependent effects at the local level as well as at a systems level.
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Interhemispheric integration of visual processing during task-driven lateralization.

TL;DR: The results provide direct neurophysiological evidence, in terms of effective connectivity, for the existence of context-dependent mechanisms of IHI that are implemented by specific visual areas during task-driven lateralization.
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Emotional processing in male adolescents with childhood-onset conduct disorder

TL;DR: Increased rather than reduced amygdala activation found in this study may indicate an enhanced response to environmental cues in adolescents with early-onset CD, and is not consistent with the assumption of a reduced capacity to take note of affective information in the social environment.
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Neurophysiological correlates of relatively enhanced local visual search in autistic adolescents

TL;DR: In this article, the authors used functional magnetic resonance imaging (fMRI) in 12 autistic adolescents (9 Asperger and 3 high-functioning autistic patients) and 12 matched controls to help distinguish, on neurophysiological grounds, between these two accounts of EFT performance in autistic patients.
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Neural basis of pantomiming the use of visually presented objects.

TL;DR: The data provide an explanation for why patients with lesions including left parietal cortex suffer from ideational apraxia as assessed by impaired object use and pantomining to visually presented objects.