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Helen Sharp

Researcher at University of Liverpool

Publications -  61
Citations -  1696

Helen Sharp is an academic researcher from University of Liverpool. The author has contributed to research in topics: Mental health & Maternal sensitivity. The author has an hindex of 18, co-authored 57 publications receiving 1457 citations. Previous affiliations of Helen Sharp include British Psychological Society & University of Leicester.

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Cognitive processes in delusional disorders.

TL;DR: This study challenges the assumption that delusional disorder (DD) patients are covertly depressed and concludes that DD is a distinct disorder predicated upon sensitivity to threat and biases of attention and attribution.
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The Leicester 500 Project. Social support and the development of postnatal depressive symptoms, a prospective cohort survey

TL;DR: Predictors of depressive symptom development differ from predictors of recovery from clinical depression in women, and interventions should be designed to reduce specific deficits in social support observed in particular study populations.
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Effects of prenatal and postnatal depression, and maternal stroking, at the glucocorticoid receptor gene

TL;DR: It is reported that GR gene (NR3C1) 1-F promoter methylation in infants is elevated in the presence of increased maternal postnatal depression following low prenatal depression, and that this effect is reversed by self-reported stroking of the infants by their mothers over the first weeks of life.
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Frequency of Infant Stroking Reported by Mothers Moderates the Effect of Prenatal Depression on Infant Behavioural and Physiological Outcomes

TL;DR: Initial findings in humans indicate that maternal stroking in infancy, as reported by mothers, has effects strongly resembling the effects of observed maternal behaviours in animals, pointing to future studies of the epigenetic, physiological and behavioral effects of maternal Stroking.
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Alternating fluency in Parkinson's disease : an evaluation of the attentional control theory of cognitive impairment

TL;DR: Parkinson's disease is associated with a deficit in inhibitory attentional processes, and an impairment in the maintenance of those internal representations which control action, which does not support the theory of internal attentional control.