H
Hidetaka Katabuchi
Researcher at Kumamoto University
Publications - 226
Citations - 6031
Hidetaka Katabuchi is an academic researcher from Kumamoto University. The author has contributed to research in topics: Ovarian cancer & Cervical cancer. The author has an hindex of 39, co-authored 218 publications receiving 5155 citations. Previous affiliations of Hidetaka Katabuchi include Johns Hopkins University.
Papers
More filters
Journal ArticleDOI
Mouse Model of Human Ovarian Endometrioid Adenocarcinoma Based on Somatic Defects in the Wnt/β-Catenin and PI3K/Pten Signaling Pathways
Rong Wu,Neali Hendrix-Lucas,Rork Kuick,Yali Zhai,Donald R. Schwartz,Aytekin Akyol,Samir M. Hanash,David E. Misek,Hidetaka Katabuchi,Bart O. Williams,Eric R. Fearon,Kathleen R. Cho +11 more
TL;DR: It is shown that defects in the PI3K/Pten and Wnt/beta-catenin signaling pathways often occur together in a subset of human OEAs, suggesting their cooperation during OEA pathogenesis.
Journal ArticleDOI
Detection of M2 macrophages and colony-stimulating factor 1 expression in serous and mucinous ovarian epithelial tumors
TL;DR: Results suggest that CSF‐1 derived from tumor tissues induces macrophages to shift toward the M2 phenotype, which is considered to promote tumor growth.
Journal ArticleDOI
Hyperintense uterine leiomyoma at T2-weighted MR imaging : differentiation with dynamic enhanced MR imaging and clinical implications
Yasuyuki Yamashita,Miyuki Torashima,Mutsumasa Takahashi,Nobuyuki Tanaka,Hidetaka Katabuchi,Kohji Miyazaki,Masaharu Ito,Hitoshi Okamura +7 more
TL;DR: Evaluation of signal intensity at T2-weighted imaging and degree of contrast enhancement at dynamic enhanced MR imaging provide useful information on the nature of leiomyomas.
Journal Article
Mutations in DNA Mismatch Repair Genes Are Not Responsible for Microsatellite Instability in Most Sporadic Endometrial Carcinomas
Hidetaka Katabuchi,Bastiaan van Rees,Anouk R. Lambers,Brigitte M. Ronnett,Marian Blazes,Fred S. Leach,Kathleen R. Cho,Lora Hedrick +7 more
TL;DR: Nine sporadic MI-positive primary endometrial carcinomas were analyzed for mutations in four known human DNA mismatch repair (MMR) genes and it was suggested that mutations in these four known DNA MMR genes are not responsible for MI in the majority of sporadic endometrian carcinomas displaying this phenotype.
Journal ArticleDOI
High-Risk Ovarian Cancer Based on 126-Gene Expression Signature Is Uniquely Characterized by Downregulation of Antigen Presentation Pathway
Kosuke Yoshihara,Tatsuhiko Tsunoda,Daichi Shigemizu,Hiroyuki Fujiwara,Masayuki Hatae,Hisaya Fujiwara,Hideaki Masuzaki,Hidetaka Katabuchi,Yosuke Kawakami,Aikou Okamoto,Takayoshi Nogawa,Noriomi Matsumura,Yasuhiro Udagawa,Tsuyoshi Saito,Hiroaki Itamochi,Masashi Takano,Etsuko Miyagi,Tamotsu Sudo,Kimio Ushijima,Haruko Iwase,Hiroyuki Seki,Yasuhisa Terao,Takayuki Enomoto,Mikio Mikami,Kohei Akazawa,Hitoshi Tsuda,Takuya Moriya,Atsushi Tajima,Ituro Inoue,Kenichi Tanaka +29 more
TL;DR: Through gene ontology and pathway analyses, a significant reduction in expression of immune-response–related genes, especially on the antigen presentation pathway, in high-risk ovarian cancer patients is identified.