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Irene M. Pedersen

Researcher at University of California, Irvine

Publications -  31
Citations -  4480

Irene M. Pedersen is an academic researcher from University of California, Irvine. The author has contributed to research in topics: microRNA & Apoptosis. The author has an hindex of 18, co-authored 31 publications receiving 4276 citations. Previous affiliations of Irene M. Pedersen include Discovery Institute & University of California, Berkeley.

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A unified model for apical caspase activation.

TL;DR: A unified caspase activation hypothesis is proposed whereby apical caspases are activated by dimerization of monomeric zymogens, and single amino acid substitutions at the dimer interface abrogate the activity of caspased-8 and -9 introduced into recipient mammalian cells.
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Interferon modulation of cellular microRNAs as an antiviral mechanism.

TL;DR: These findings strongly support the notion that mammalian organisms too, through the interferon system, use cellular miRNAs to combat viral infections.
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Small-molecule antagonists of apoptosis suppressor XIAP exhibit broad antitumor activity.

TL;DR: An enzyme derepression assay is developed, based on overcoming XIAP-mediated suppression of Caspase-3, and mixture-based combinatorial chemical libraries are screened for compounds that reversed XI AP-mediated inhibition of Cazase- 3, identifying a class of polyphenylureas with XIAP -inhibitory activity.
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Src homology 2 domain–containing inositol-5-phosphatase and CCAAT enhancer-binding protein β are targeted by miR-155 in B cells of Eμ-MiR-155 transgenic mice

TL;DR: It is hypothesized that miR-155, by down-modulating Ship and C/EBPbeta, initiates a chain of events that leads to the accumulation of large pre-B cells and acute lymphoblastic leukemia/high-grade lymphoma.
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Protection of CLL B cells by a follicular dendritic cell line is dependent on induction of Mcl-1.

TL;DR: HK cells protect CLL B cells against apoptosis, at least in part through a CD44-dependent mechanism involving up-regulation of Mcl-1, and this mechanism is distinct from that achieved by CD40 ligation.