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John K. Robinson

Researcher at Stony Brook University

Publications -  80
Citations -  3932

John K. Robinson is an academic researcher from Stony Brook University. The author has contributed to research in topics: Galanin & Neuropeptide. The author has an hindex of 32, co-authored 79 publications receiving 3553 citations. Previous affiliations of John K. Robinson include State University of New York System & University of New Hampshire.

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A complement–microglial axis drives synapse loss during virus-induced memory impairment

TL;DR: It is shown that viral infection of adult hippocampal neurons induces complement-mediated elimination of presynaptic terminals in a murine WNV neuroinvasive disease model, which provides a new murine model of WNV-induced spatial memory impairment, and identifies a potential mechanism underlying neurocognitive impairment in patients recovering from WNV Neuroinvasive Disease.
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Anandamide, an endogenous ligand of the cannabinoid receptor, induces hypomotility and hypothermia in vivo in rodents

TL;DR: It is demonstrated that anandamide has biological and behavioral effects in awake rodents, some of which are similar to the reported actions of THC.
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Exercise influences spatial learning in the radial arm maze.

TL;DR: Voluntarily exercising rats ran in running wheels attached to their home cage for 7 weeks prior to and throughout testing, and took 30% fewer trials to acquire criterion performance than sedentary controls, but group differences were not apparent.
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Minocycline Reduces Microglial Activation and Improves Behavioral Deficits in a Transgenic Model of Cerebral Microvascular Amyloid

TL;DR: It is suggested that anti-inflammatory treatment targeted for cerebral microvascular amyloid-induced microglial activation can improve cognitive deficits without altering the accumulation and distribution of Aβ.
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MeCP2 Is Critical for Maintaining Mature Neuronal Networks and Global Brain Anatomy during Late Stages of Postnatal Brain Development and in the Mature Adult Brain

TL;DR: It is shown that brains that lose MeCP2 at these two different stages are actively shrinking, resulting in higher than normal neuronal cell density and that mature dendritic arbors of pyramidal neurons are severely retracted and d endritic spine density is dramatically reduced.