J
Jonathan Ellis
Researcher at Queensland University of Technology
Publications - 41
Citations - 2362
Jonathan Ellis is an academic researcher from Queensland University of Technology. The author has contributed to research in topics: Exome sequencing & Population. The author has an hindex of 20, co-authored 38 publications receiving 1996 citations. Previous affiliations of Jonathan Ellis include University of Queensland & Princess Alexandra Hospital.
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Journal ArticleDOI
Calypso: a user-friendly web-server for mining and visualizing microbiome–environment interactions
Martha Zakrzewski,Carla Proietti,Jonathan Ellis,Shihab Hasan,Shihab Hasan,Marie-Jo Brion,Bernard Berger,Lutz Krause,Lutz Krause +8 more
TL;DR: Calypso is an easy‐to‐use online software suite that allows non‐expert users to mine, interpret and compare taxonomic information from metagenomic or 16S rDNA datasets and has a focus on multivariate statistical approaches that can identify complex environment‐microbiome associations.
Journal ArticleDOI
Molecular basis for specificity of nuclear import and prediction of nuclear localization
Mary Marfori,Andrew V. Mynott,Jonathan Ellis,Ahmed M. Mehdi,Neil F. W. Saunders,Paul M. G. Curmi,Jade K. Forwood,Mikael Bodén,Bostjan Kobe,Bostjan Kobe +9 more
TL;DR: In this paper, the authors review the current understanding of the molecular determinants of the specificity of nuclear import, focusing on the importin-α•cargo recognition, as well as the currently available databases and predictive tools relevant to nuclear localization.
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Protein-RNA Interactions: structural analysis and functional classes
TL;DR: The analysis of hydrogen bond and van der Waal contacts showed that in general proteins complexed with messenger RNA, transfer RNA and viral RNA have more base specific contacts and less backbone contacts than expected, while proteins complexing with ribosomal RNA have less base‐specific contacts than the expected.
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Frequent somatic mutations in MAP3K5 and MAP3K9 in metastatic melanoma identified by exome sequencing
Mitchell S. Stark,Susan L. Woods,Michael Gartside,Vanessa F. Bonazzi,Ken Dutton-Regester,Ken Dutton-Regester,Lauren G. Aoude,Lauren G. Aoude,Donald Chow,Chris Sereduk,Natalie M. Niemi,Nanyun Tang,Jonathan Ellis,Jeffrey S. Reid,Victoria Zismann,Sonika Tyagi,Donna M. Muzny,Irene Newsham,Yuanqing Wu,Jane M. Palmer,Thomas Pollak,David Youngkin,Bradford R. Brooks,Catherine M. Lanagan,Christopher W. Schmidt,Bostjan Kobe,Jeffrey P. MacKeigan,Hongwei Yin,Kevin M. Brown,Kevin M. Brown,Richard A. Gibbs,Jeffrey M. Trent,Jeffrey M. Trent,Nicholas K. Hayward +33 more
TL;DR: Attenuation of MAP3K9 function in melanoma cells using siRNA led to increased cell viability after temozolomide treatment, suggesting that decreasedMAP3K pathway activity can lead to chemoresistance in melanomas.
Journal ArticleDOI
Smchd1 regulates a subset of autosomal genes subject to monoallelic expression in addition to being critical for X inactivation
Arne W. Mould,Arne W. Mould,Zhenyi Pang,Miha Pakusch,Ian D. Tonks,Mitchell S. Stark,Dianne Carrie,Pamela Mukhopadhyay,Annica Seidel,Jonathan Ellis,Janine E. Deakin,Matthew Wakefield,Matthew Wakefield,Lutz Krause,Marnie E. Blewitt,Marnie E. Blewitt,Graham F. Kay +16 more
TL;DR: These findings, combined with the recent report implicating heterozygous mutations of SMCHD1 as a causal factor in the digenically inherited muscular weakness syndrome facioscapulohumeral muscular dystrophy-2, highlight the potential importance of Smchd1 in the etiology of diverse human diseases.