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Kimihiro Komori

Researcher at Nagoya University

Publications -  219
Citations -  5176

Kimihiro Komori is an academic researcher from Nagoya University. The author has contributed to research in topics: Abdominal aortic aneurysm & Endovascular aneurysm repair. The author has an hindex of 32, co-authored 207 publications receiving 4170 citations. Previous affiliations of Kimihiro Komori include Kyushu University.

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Global vascular guidelines on the management of chronic limb-threatening ischemia

Michael S. Conte, +58 more
TL;DR: The GVG proposes a new Global Anatomic Staging System (GLASS), which involves defining a preferred target artery path (TAP) and then estimating limb-based patency (LBP) resulting in three stages of complexity for intervention.
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Angiogenic gene therapy for experimental critical limb ischemia: Acceleration of limb loss by overexpression of vascular endothelial growth factor 165 but not of fibroblast growth factor-2

TL;DR: VEGF is necessary, but should be delicately regulated to lower expression to treat ischemic limb, and the therapeutic effect of FGF-2, associated with the harmonized angiogenic effects seen with endogenous VEGF, provides important insights into therapeutic angiogenesis.
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Electrical responses of smooth muscle cells during cholinergic vasodilation in the rabbit saphenous artery.

TL;DR: The results suggest that the inhibitory actions of muscarinic agonists on electrical responses of smooth muscle cells of the rabbit saphenous artery were mainly indirect, i.e., a release of inhibitory substances from the endothelial cells and the inhibition of adrenerglc transmission.
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Essential role of monocyte chemoattractant protein-1 in development of restenotic changes (neointimal hyperplasia and constrictive remodeling) after balloon angioplasty in hypercholesterolemic rabbits.

TL;DR: MCP-1–mediated monocyte infiltration is necessary in the development of restenotic changes to balloon injury in hypercholesterolemic rabbits and this strategy may be a useful and practical form of gene therapy against human restenosis.