L
Lance D. Miller
Researcher at Wake Forest University
Publications - 208
Citations - 16169
Lance D. Miller is an academic researcher from Wake Forest University. The author has contributed to research in topics: Cancer & Breast cancer. The author has an hindex of 58, co-authored 195 publications receiving 14459 citations. Previous affiliations of Lance D. Miller include University of Texas MD Anderson Cancer Center & East Carolina University.
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Journal ArticleDOI
Immune signature in sarcoma with prognostic and predictive implications.
Shailaja Ks Raj,Lance D. Miller,Melissa Winters,Gregory B. Russell,Triozzi Pierre,Paul D. Savage +5 more
TL;DR: This study sought to evaluate the efficacy of immunotherapy in sarcoma by evaluating its benefit in the management of many solid tumors and its safety and tolerability in patients with high Mayer-Leroy syndrome.
Proceedings ArticleDOI
Abstract 4715: Protective anti-tumor immune responses in breast cancer depend on tumor mutation rate
Proceedings ArticleDOI
Multimodality as a criterion for feature selection in unsupervised analysis of gene expression data
TL;DR: The use of multimodality as a criterion for choosing genes in feature selection is examined, and a novel measure of pairwise dissimilarity is proposed to cluster the genes that have survived the preprocessing step.
Posted ContentDOI
NuMA is a negative regulator of 53BP1 in DNA double-strand break repair
Naike Salvador Moreno,Jing Liu,Karen M. Haas,Laurie L. Parker,Chaitali Chakraborty,Stephen J. Kron,Kurt Hodges,Lance D. Miller,Paul J. Robinson,Sophie A. Lelièvre,Pierre-Alexandre Vidi +10 more
TL;DR: Regulation of 53BP1 beyond repair foci is shown and this mechanism may have evolved to disable repair functions and may be a decisive factor for tumor responses to genotoxic treatments.
Proceedings ArticleDOI
Abstract 78: Fractionated whole brain irradiation modulates Homer1a expression in a brain region specific manner.
TL;DR: Fractionated whole brain irradiation modulates Homer1a expression in a brain region specific manner, and memory/learning paradigms suggest that trafficking of 2-amino-3-(3-hydroxy-5-methyl-isoxazol-4-yl)propanoic acid (AMPA) receptors containing mGluR1 from intracellular compartments to the plasma membrane is an “early” event during neural plasticity; thus decreased mGle