Laura Dindia

TL;DR: The classic heat shock (stress) response (HSR) was originally attributed to protein denaturation, but it is observed that a deregulated HSR is found in a large number of important diseases where more detailed knowledge of the molecular mechanisms involved may offer timely opportunities for clinical interventions and new, innovative drug treatments.

TL;DR: Results indicate that stressor-induced elevation of plasma cortisol level is associated with alterations in plasma membrane fluidity and rapid activation of stress-related signaling pathways in trout liver.

TL;DR: In this paper, stress levels of cortisol, the primary glucocorticoid in teleosts, rapidly fluidizes rainbow trout (Oncorhynchus mykiss) liver plasma membranes in vitro.

TL;DR: It is shown that stress levels of cortisol rapidly fluidizes rainbow trout (Oncorhynchus mykiss) liver plasma membranes in vitro, suggesting that biophysical changes to plasma membrane properties, triggered by stressor-induced glucocorticoid elevation, act as a nonspecific stress response and may rapidly modulate acute stress-signaling pathways.

TL;DR: In this paper, the authors present a workshop on pre-lecture activities to help students prepare for lectures and provide a basis for interactive learning, without compromising the amount of content that can be taught during the lecture.