M
M. Imad Damaj
Researcher at Virginia Commonwealth University
Publications - 176
Citations - 5593
M. Imad Damaj is an academic researcher from Virginia Commonwealth University. The author has contributed to research in topics: Nicotine & Nicotinic agonist. The author has an hindex of 38, co-authored 161 publications receiving 4791 citations. Previous affiliations of M. Imad Damaj include VCU Medical Center & St. Joseph's Hospital and Medical Center.
Papers
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Journal ArticleDOI
Guidelines on nicotine dose selection for in vivo research
Shannon G. Matta,David J.K. Balfour,Neal L. Benowitz,R. Thomas Boyd,Jerry J. Buccafusco,Anthony R. Caggiula,Caroline R. Craig,Allan C. Collins,M. Imad Damaj,Eric C. Donny,Phillip S. Gardiner,Sharon R. Grady,Ulrike Heberlein,Sherry Leonard,Edward D. Levin,Ronald J. Lukas,Athina Markou,Michael J. Marks,Sarah E. McCallum,Neeraja Parameswaran,Kenneth A. Perkins,Marina R. Picciotto,Maryka Quik,Jed E. Rose,Adrian Rothenfluh,William R Schafer,Ian P. Stolerman,Rachel F. Tyndale,Jeanne M. Wehner,Jeffrey M. Zirger +29 more
TL;DR: A new, experimentally based compilation of species-specific dose selection for studies on the in vivo effects of nicotine, addressing issues related to genetic background, age, acute vs chronic exposure, route of administration, and behavioral responses is provided.
Journal ArticleDOI
Enantioselective effects of hydroxy metabolites of bupropion on behavior and on function of monoamine transporters and nicotinic receptors.
M. Imad Damaj,F. Ivy Carroll,J. Brek Eaton,Hernán A. Navarro,Bruce E. Blough,Sadiq Mirza,Ronald J. Lukas,Billy R. Martin +7 more
TL;DR: The results suggest that clinical and behavioral effects of bupropion arise from actions at nAChR as well as DA and NE transporters and that the (2S,3S)-hydroxybupropion isomer may be a better drug candidate for smoking cessation because of its higher potency at the relevant targets.
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The β2 but not α7 subunit of the nicotinic acetylcholine receptor is required for nicotine-conditioned place preference in mice
TL;DR: Data suggest that the β2 subunit of the nAChR is critically involved in nicotine reward as measured by CPP, which is similar to that shown in wild-type mice.
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The antinociceptive effects of α7 nicotinic agonists in an acute pain model
TL;DR: These studies suggest that activation of α 7 receptors in the CNS elicits antinociceptive effects in an acute thermal pain model.
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Early Smoking Onset and Risk for Subsequent Nicotine Dependence: A Monozygotic Co-Twin Control Study
TL;DR: Findings suggest that in humans, early nicotine exposure directly increases level of later nicotine dependence, consistent with the animal literature.