M
Maja Olsson
Researcher at Nanyang Technological University
Publications - 26
Citations - 1253
Maja Olsson is an academic researcher from Nanyang Technological University. The author has contributed to research in topics: Adipose tissue & Serum amyloid A. The author has an hindex of 12, co-authored 23 publications receiving 766 citations. Previous affiliations of Maja Olsson include University of Gothenburg & Lund University.
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Journal ArticleDOI
The humanistic and economic burden of chronic wounds: A systematic review.
Maja Olsson,Krister Järbrink,Ushashree Divakar,Ram Bajpai,Zee Upton,Zee Upton,Artur Schmidtchen,Artur Schmidtchen,Josip Car,Josip Car +9 more
TL;DR: A systematic review of chronic wounds revealed that health‐related quality of life was lowest for physical pathologies, and based on average estimates were scores most inferior in the domain physical role for both patients with chronic wounds and for those with wound‐related amputations.
Journal ArticleDOI
Prevalence of chronic wounds in the general population: systematic review and meta-analysis of observational studies.
Laura Martinengo,Maja Olsson,Ram Bajpai,Michael Soljak,Zee Upton,Artur Schmidtchen,Josip Car,Josip Car,Krister Järbrink +8 more
TL;DR: The findings showed that the vast majority of chronic wounds in epidemiological studies are made up by chronic leg ulcers, which is aligned to previous studies reporting point prevalence of chronic injuries identified within the healthcare system.
Journal ArticleDOI
Improving the pathogenicity of a nematode-trapping fungus by genetic engineering of a subtilisin with nematotoxic activity.
Johan Åhman,Tomas Johansson,Maja Olsson,Peter J. Punt,C.A.M.J.J. van den Hondel,Anders Tunlid +5 more
TL;DR: It is reported that a subtilisin-like extracellular serine protease designated PII is an important pathogenicity factor in the common nematode-trapping fungus Arthrobotrys oligospora and the paralyzing activity of PII was verified by demonstrating that a heterologous-produced PII had a nematotoxic activity when added to free-living nematodes.
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Genome-wide association meta-analysis of functional outcome after ischemic stroke
Martin Söderholm,Annie Pedersen,Erik Lorentzen,Tara M. Stanne,Steve Bevan,Maja Olsson,John W. Cole,Israel Fernandez-Cadenas,Israel Fernandez-Cadenas,Graeme J. Hankey,Jordi Jimenez-Conde,Jordi Jimenez-Conde,Katarina Jood,Jin-Moo Lee,Robin Lemmens,Robin Lemmens,Robin Lemmens,Christopher Levi,Christopher Levi,Braxton D. Mitchell,Bo Norrving,Kristiina Rannikmäe,Natalia S. Rost,Jonathan Rosand,Jonathan Rosand,Jonathan Rosand,Peter M. Rothwell,Rodney Scott,Rodney Scott,Daniel Strbian,Jonathan Sturm,Cathie Sudlow,Matthew Traylor,Vincent Thijs,Vincent Thijs,Turgut Tatlisumak,Turgut Tatlisumak,Daniel Woo,Bradford B. Worrall,Jane Maguire,Arne Lindgren,Christina Jern,Christina Jern +42 more
TL;DR: This large GWA study on functional outcome after ischemic stroke reports one significant variant and several variants with suggestive association to outcome 3 months after stroke onset with plausible mechanistic links to poststroke recovery.
Journal ArticleDOI
Expression of the selenoprotein S (SELS) gene in subcutaneous adipose tissue and SELS genotype are associated with metabolic risk factors.
Maja Olsson,Bob Olsson,Peter Jacobson,Dag S. Thelle,Dag S. Thelle,Johan Björkegren,Andrew Walley,Philippe Froguel,Philippe Froguel,Lena M. S. Carlsson,Kajsa Sjöholm +10 more
TL;DR: A role for SELS in the development of metabolic disease, especially in the context of insulin resistance is supported, and in vitro studies demonstrated that the SELS gene is regulated by insulin in human subcutaneous adipocytes.