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Maja Olsson

Researcher at Nanyang Technological University

Publications -  26
Citations -  1253

Maja Olsson is an academic researcher from Nanyang Technological University. The author has contributed to research in topics: Adipose tissue & Serum amyloid A. The author has an hindex of 12, co-authored 23 publications receiving 766 citations. Previous affiliations of Maja Olsson include University of Gothenburg & Lund University.

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The humanistic and economic burden of chronic wounds: A systematic review.

TL;DR: A systematic review of chronic wounds revealed that health‐related quality of life was lowest for physical pathologies, and based on average estimates were scores most inferior in the domain physical role for both patients with chronic wounds and for those with wound‐related amputations.
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Prevalence of chronic wounds in the general population: systematic review and meta-analysis of observational studies.

TL;DR: The findings showed that the vast majority of chronic wounds in epidemiological studies are made up by chronic leg ulcers, which is aligned to previous studies reporting point prevalence of chronic injuries identified within the healthcare system.
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Improving the pathogenicity of a nematode-trapping fungus by genetic engineering of a subtilisin with nematotoxic activity.

TL;DR: It is reported that a subtilisin-like extracellular serine protease designated PII is an important pathogenicity factor in the common nematode-trapping fungus Arthrobotrys oligospora and the paralyzing activity of PII was verified by demonstrating that a heterologous-produced PII had a nematotoxic activity when added to free-living nematodes.
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Genome-wide association meta-analysis of functional outcome after ischemic stroke

TL;DR: This large GWA study on functional outcome after ischemic stroke reports one significant variant and several variants with suggestive association to outcome 3 months after stroke onset with plausible mechanistic links to poststroke recovery.
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Expression of the selenoprotein S (SELS) gene in subcutaneous adipose tissue and SELS genotype are associated with metabolic risk factors.

TL;DR: A role for SELS in the development of metabolic disease, especially in the context of insulin resistance is supported, and in vitro studies demonstrated that the SELS gene is regulated by insulin in human subcutaneous adipocytes.