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Natalia S. Rost

Researcher at Harvard University

Publications -  311
Citations -  12393

Natalia S. Rost is an academic researcher from Harvard University. The author has contributed to research in topics: Stroke & Medicine. The author has an hindex of 50, co-authored 259 publications receiving 9279 citations. Previous affiliations of Natalia S. Rost include Joseph Fourier University & Brigham and Women's Hospital.

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Plasma Concentration of C-Reactive Protein and Risk of Ischemic Stroke and Transient Ischemic Attack: The Framingham Study

TL;DR: Elevated plasma CRP levels significantly predict the risk of future ischemic stroke and TIA in the elderly, independent of other cardiovascular risk factors.

Multiancestry genome-wide association study of 520,000 subjects identifies 32 loci associated with stroke and stroke subtypes

Rainer Malik, +170 more
TL;DR: A multiancestry genome-wide-association meta-analysis and discovered 22 new stroke risk loci, indicating mechanisms not previously implicated in stroke pathophysiology, with prioritization of risk variants and genes accomplished through bioinformatics analyses using extensive functional datasets.
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Multi-Ethnic Genome-wide Association Study for Atrial Fibrillation

Carolina Roselli, +260 more
- 11 Jun 2018 - 
TL;DR: This large, multi-ethnic genome-wide association study identifies 97 loci significantly associated with atrial fibrillation that are enriched for genes involved in cardiac development, electrophysiology, structure and contractile function.
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Prediction of Functional Outcome in Patients With Primary Intracerebral Hemorrhage The FUNC Score

TL;DR: Func score is a valid clinical assessment tool that identifies patients with ICH who will attain functional independence and thus, can provide guidance in clinical decision-making and patient selection for clinical trials.
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Aspirin and recurrent intracerebral hemorrhage in cerebral amyloid angiopathy

TL;DR: Recurrence of lobar ICH is associated with previous microbleeds or macrobleeds and posterior CT white matter hypodensity, which may be markers of severity for underlying cerebral amyloid angiopathy.