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Maria Rosa Chitolina Schetinger

Researcher at Universidade Federal de Santa Maria

Publications -  470
Citations -  10620

Maria Rosa Chitolina Schetinger is an academic researcher from Universidade Federal de Santa Maria. The author has contributed to research in topics: TBARS & Oxidative stress. The author has an hindex of 44, co-authored 447 publications receiving 9147 citations. Previous affiliations of Maria Rosa Chitolina Schetinger include Universidade Federal do Rio Grande do Sul.

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Dietary Supplementation of Ginger and Turmeric Rhizomes Modulates Platelets Ectonucleotidase and Adenosine Deaminase Activities in Normotensive and Hypertensive Rats

TL;DR: Dairy supplementation with turmeric or ginger efficiently prevented platelet alterations by modulating the hydrolysis of ATP, ADP and AMP with a concomitant decrease in ADA activity, which could suggest some possible mechanism of the rhizomes against hypertension‐derived complications associated to platelet hyperactivity.
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Effect of subchronic treatment with mercury chloride on NTPDase, 5'-nucleotidase and acetylcholinesterase from cerebral cortex of rats.

TL;DR: It is shown for the first time that exposure to low levels of Hg2+, which resembles occupational exposure toLow levels of mercury, caused a marked increase in NTPDase and AChE activities and the relationship of these alterations with the neurotoxicity of inorganic mercury deserves further studies.
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Effect of high glucose levels in human platelet NTPDase and 5′-nucleotidase activities

TL;DR: The hydrolysis of adenine nucleotides is enhanced in platelets of patients with diabetes and hypertension as observed in NTPDase and 5'-nucleotidase activities, indicating that these enzyme activities are related with Diabetes and hypertension.
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Adenosine signaling and adenosine deaminase regulation of immune responses: impact on the immunopathogenesis of HIV infection

TL;DR: The connection between adenosinergic system and HIV immunopathogenesis is explored, exploring defects in immune cell function and the role of ADA in protecting these cells against damage.