scispace - formally typeset
M

Maria Svelto

Researcher at University of Bari

Publications -  197
Citations -  8331

Maria Svelto is an academic researcher from University of Bari. The author has contributed to research in topics: Aquaporin & Water transport. The author has an hindex of 52, co-authored 188 publications receiving 7471 citations. Previous affiliations of Maria Svelto include University of Basilicata.

Papers
More filters
Journal ArticleDOI

Analysis by two-dimensional Blue Native/SDS-PAGE of membrane protein alterations in rat soleus muscle after hindlimb unloading

TL;DR: The alterations of soleus muscle membrane proteome may play a pivotal role in the mechanisms involved in disuse-induced muscle atrophy.
Journal ArticleDOI

Aquaporin-1 expression in the chick embryo chorioallantoic membrane

TL;DR: Investigation of developmental expression of AQP1 in the chick CAM by Western blot and immunohistochemistry shows that a major nonglycosylated band was observed with electrophoretic mobility in the three developmental stages examined, implying a stimulation or an inhibition of angiogenesis in the CAM assay.
Journal ArticleDOI

Hepatocyte and Sertoli Cell Aquaporins, Recent Advances and Research Trends

TL;DR: This review focuses on the most recent advances and research trends regarding the expression and modulation, as well as physiological and pathophysiological functions of AQPs in hepatocytes and Sertoli cells (SCs).
Journal ArticleDOI

In vivo silencing of aquaporin-1 by RNA interference inhibits angiogenesis in the chick embryo chorioallantoic membrane assay.

TL;DR: The hypothesis that AQP1 could have a key role in physiological and pathological angiogenesis is supported by the first study showing the in vivo use of RNA interference technique in the CAM assay.
Journal ArticleDOI

Altered expression of renal aquaporins and α-adducin polymorphisms may contribute to the establishment of salt-sensitive hypertension.

TL;DR: First evidence that AQP1, AQP2, and AQP4 are dysregulated in the kidneys of MHS animals is provided and evidence that α-adducin mutations may be responsible for AQP 4 upregulation is provided.