M
Martin Berry
Researcher at University of Birmingham
Publications - 325
Citations - 16166
Martin Berry is an academic researcher from University of Birmingham. The author has contributed to research in topics: Axon & Optic nerve. The author has an hindex of 67, co-authored 324 publications receiving 15711 citations. Previous affiliations of Martin Berry include Centre national de la recherche scientifique & University of Bristol.
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Role of the two activating domains of the oestrogen receptor in the cell-type and promoter-context dependent agonistic activity of the anti-oestrogen 4-hydroxytamoxifen.
TL;DR: It is demonstrated that the agonistic effect of OHT on the whole hER is due to the cell‐type and promoter‐context dependent activity of TAF‐1, and it is confirmed that the wild type human oestrogen receptor has no ligand independent transcriptional activity.
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Myelin-, reactive glia-, and scar-derived CNS axon growth inhibitors: expression, receptor signaling, and correlation with axon regeneration
TL;DR: Axon regeneration is arrested in the injured central nervous system (CNS) by axon growth-inhibitory ligands expressed in oligodendrocytes/myelin, NG2-glia, and reactive astrocyte in the lesion and degenerating tracts, and by fibroblasts in scar tissue.
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A versatile reducible polycation-based system for efficient delivery of a broad range of nucleic acids
Martin L. Read,Surjeet Singh,Zubair Ahmed,Mark Stevenson,Simon S. Briggs,David Oupicky,Lee Barrett,Rachel Spice,Mark A. F. Kendall,Martin Berry,Jon A. Preece,Ann Logan,Leonard W. Seymour +12 more
TL;DR: Results demonstrate that HIS RPCs represent a novel and versatile type of vector that can be used for efficient cytoplasmic delivery of a broad range of nucleic acids.
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Observations on the astrocyte response to a cerebral stab wound in adult rats
TL;DR: It is proposed that astrocytes respond primarily to the mechanical disruption consequent to injury and that the response promotes the restoration of the structural integrity of the lesioned tissue.
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Fibroblast growth factors promote the survival of adult rat retinal ganglion cells after transection of the optic nerve
TL;DR: Results indicate that FGF has neurotrophic activity in the adult central nervous system, and that this molecule is able to rescue adult retinal ganglion cells from axotomy induced cell death.