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Masashi Mizokami

Researcher at Nagoya City University

Publications -  684
Citations -  37168

Masashi Mizokami is an academic researcher from Nagoya City University. The author has contributed to research in topics: Hepatitis B virus & Hepatitis C virus. The author has an hindex of 85, co-authored 650 publications receiving 34868 citations. Previous affiliations of Masashi Mizokami include National Institute of Advanced Industrial Science and Technology & University of Florida.

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Lack of association between TTV viral load and aminotransferase levels in patients with hepatitis C or non-B-C.

TL;DR: In cases of chronic hepatitis, no correlation was observed between the serum TTV DNA titres and the ALT levels, and in 2 patients who were treated with interferon, the changes in TTV titres were not synchronized with those of theALT levels.
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Identification and phylogenetic analysis of hepatitis C virus in forensic blood samples obtained from injecting drug users.

TL;DR: The transmission of HCV between IDUs was demonstrated, and this indicates that phylogenetic analysis would applicable to also forensic analysis, thus multiple transmission route of HCv among IDUs seems be exist in Japan.
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Easy-to-use phylogenetic analysis system for hepatitis B virus infection

TL;DR: The phylogenetic analysis tool was developed for the easy determination of genotypes and transmission route and the appropriate settings and application of calculation parameters are difficult for non‐specialists of molecular genetics.
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Characterization of infectious hepatitis C virus from liver-derived cell lines.

TL;DR: The efficient production of infectious HCV from the JFH-1 strain is restricted to the Huh7 cell line and its derivatives, but the virus particles produced from the different cell lines may have different characteristics.
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Negative regulation of hepatitis B virus replication by forkhead box protein A in human hepatoma cells.

TL;DR: It is found that FOXA members are directly and indirectly involved in HBV replication in human hepatic cells and FoxA1 and FOXA2 suppressed nuclear hormone receptors, such as HNF4α, that are related toHBV replication.