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Institution

Toray Industries

CompanyNorth Kingstown, Rhode Island, United States
About: Toray Industries is a company organization based out in North Kingstown, Rhode Island, United States. It is known for research contribution in the topics: Layer (electronics) & Fiber. The organization has 6134 authors who have published 5746 publications receiving 86632 citations. The organization is also known as: Toray & Toray Industries, Inc..
Topics: Layer (electronics), Fiber, Polyester, Membrane, Epoxy


Papers
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Journal ArticleDOI
TL;DR: In this paper, the authors demonstrate that PBDTT-DPP, a semiconducting polymer with a low bandgap of 1.44 eV, allows tandem polymer solar cells to reach power conversion efficiencies of around 8.6%.
Abstract: Researchers demonstrate that PBDTT-DPP, a semiconducting polymer with a low bandgap of 1.44 eV, allows tandem polymer solar cells to reach power conversion efficiencies of around 8.6%.

1,406 citations

Journal ArticleDOI
08 Apr 1994-Cell
TL;DR: The purification and cloning of APRF are reported and it is observed that p91 is not tyrosine phosphorylated in response to IL-6, and that selective activation of p91-related factors may explain the diversity of cellular responses to different cytokines.

997 citations

Journal ArticleDOI
TL;DR: The results demonstrate that the amyloids associated with chronic hemodialysis contains as major component a new form of amyloid fibril protein that is homologous to beta 2-microglobulin.

961 citations

Journal ArticleDOI
18 Apr 2002-Nature
TL;DR: This study shows that RANKL induces the interferon-β (IFN- β) gene in osteoclast precursor cells, and that IFN-β inhibits the differentiation by interfering with the RankL-induced expression of c-Fos, an essential transcription factor for the formation of osteoclasts.
Abstract: Osteoclasts are cells of monocyte/macrophage origin that erode bone matrix: regulation of their differentiation is central to the understanding of the pathogenesis and treatment of bone diseases such as osteoporosis. Signalling by RANKL (receptor activator of NF-kappaB ligand), also known as Tnfsf11, is essential for the induction of osteoclast differentiation, and it must be strictly regulated to maintain bone homeostasis. But it is not known whether RANKL signalling to the cell interior is linked to any regulatory mechanisms. Here we show that RANKL induces the interferon-beta (IFN-beta) gene in osteoclast precursor cells, and that IFN-beta inhibits the differentiation by interfering with the RANKL-induced expression of c-Fos, an essential transcription factor for the formation of osteoclasts. This IFN-beta gene induction mechanism is distinct from that induced by virus, and is dependent on c-Fos itself. Thus an autoregulatory mechanism operates-the RANKL-induced c-Fos induces its own inhibitor. The importance of this regulatory mechanism for bone homeostasis is emphasized by the observation that mice deficient in IFN-beta signalling exhibit severe osteopenia (loss of bone mass) accompanied by enhanced osteoclastogenesis. Our study places the IFN-beta system in a new context, and may offer a molecular basis for the treatment of bone diseases.

682 citations


Authors

Showing all 6142 results

NameH-indexPapersCitations
Tadatsugu Taniguchi12335869132
Gerald J. Gleich10860646698
Atsushi Ochiai8154724887
Ken-ichi Inui7838820017
Raymond M. Hakim7721418084
Koichi Suzuki7329317868
Hisanori Shinohara7263720020
Hirohito Kita7028316823
Osamu Ogawa7073819302
Taka-hisa Arima6742625692
Isao Mochida6474218355
Takahiro Ochiya6430919828
Enyu Imai6233918121
Yoshitaka Isaka5637919611
Yoshiharu Sakai5342811525
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Performance
Metrics
No. of papers from the Institution in previous years
YearPapers
20237
20223
202142
2020152
2019225
2018186